Inflammatory signaling pathways induced by Helicobacter pylori in primary human gastric epithelial cells.

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Confirming the Effects of Qinghuayin against Chronic Atrophic Gastritis and a Preliminary Observation of the Involved Inflammatory Signaling Pathways: An In Vivo Study

P2860

Q57821266-C476E6B5-546B-4AE9-ADD9-6535B0B63D91

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Inflammatory signaling pathways induced by Helicobacter pylori in primary human gastric epithelial cells.

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http://www.wikidata.org/entity/Q38727805

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2016 nî lūn-bûn

@nan

2016年の論文

@ja

2016年論文

@yue

2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

@zh-tw

2016年论文

@wuu

2016年论文

@zh

2016年论文

@zh-cn

name

Inflammatory signaling pathway ...... uman gastric epithelial cells.

@en

type

Item

label

Inflammatory signaling pathway ...... uman gastric epithelial cells.

@en

prefLabel

Inflammatory signaling pathway ...... uman gastric epithelial cells.

@en

P1476

Inflammatory signaling pathway ...... human gastric epithelial cells

@en

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Christophe Burucoa

http://www.w3.org/2001/XMLSchema#string

Martine Garnier

http://www.w3.org/2001/XMLSchema#string

P2860

Pathogenesis of Helicobacter pylori infection

Interplay of the Gastric Pathogen Helicobacter pylori with Toll-Like Receptors

Toll-like receptor (TLR) 2 and TLR5, but not TLR4, are required for Helicobacter pylori-induced NF-kappa B activation and chemokine expression by epithelial cells

Helicobacter pylori induces interleukin-8 secretion by Toll-like receptor 2- and Toll-like receptor 5-dependent and -independent pathways

NF-kappaB activation and potentiation of proinflammatory responses by the Helicobacter pylori CagA protein.

Induction of TLR-2 and TLR-5 expression by Helicobacter pylori switches cagPAI-dependent signalling leading to the secretion of IL-8 and TNF-α.

Helicobacter pylori versus the host: remodeling of the bacterial outer membrane is required for survival in the gastric mucosa.

Interleukin-8 is the single most up-regulated gene in whole genome profiling of H. pylori exposed gastric epithelial cells.

The Helicobacter pylori urease B subunit binds to CD74 on gastric epithelial cells and induces NF-kappaB activation and interleukin-8 production.

Nod1 responds to peptidoglycan delivered by the Helicobacter pylori cag pathogenicity island.

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165-174

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scholarly article

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10.1177/1753425916681077

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Magali Garcia

Charles Bodet

Cong Tri Tran

P577

2016-12-05T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

27913793

http://www.w3.org/2001/XMLSchema#string

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Helicobacter pylori

Inflammatory signaling pathways induced by Helicobacter pylori in primary human gastric epithelial cells.

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P2860

P2860

Inflammatory signaling pathways induced by Helicobacter pylori in primary human gastric epithelial cells.

description

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type

label

prefLabel

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P698

P921

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P698

P921