Tissue mechanics promote IDH1-dependent HIF1α-tenascin C feedback to regulate glioblastoma aggression. - Wikidata - wikimedia - TriplyDB

Tissue mechanics promote IDH1-dependent HIF1α-tenascin C feedback to regulate glioblastoma aggression.

Tissue mechanics promote IDH1-dependent HIF1α-tenascin C feedback to regulate glioblastoma aggression.

http://www.wikidata.org/entity/Q38732546

about

Mechanical characterization of human brain tumors from patients and comparison to potential surgical phantoms The Role of Tumor Microenvironment in Chemoresistance: To Survive, Keep Your Enemies Closer.Tissue mechanics regulate brain development, homeostasis and disease.The Microenvironmental Landscape of Brain Tumors.Physical and Chemical Gradients in the Tumor Microenvironment Regulate Tumor Cell Invasion, Migration, and Metastasis.Quantifying forces in cell biology.Emerging roles of mechanical forces in chromatin regulation.Modeling tumor cell adaptations to hypoxia in multicellular tumor spheroids Transcriptional response to hypoxic stress in melanoma and prognostic potential of GBE1 and BNIP3.Soft Substrates Containing Hyaluronan Mimic the Effects of Increased Stiffness on Morphology, Motility, and Proliferation of Glioma Cells.Brain-Mimetic 3D Culture Platforms Allow Investigation of Cooperative Effects of Extracellular Matrix Features on Therapeutic Resistance in Glioblastoma.MR Elastography Analysis of Glioma Stiffness and IDH1-Mutation Status.The Influence of Hyaluronic Acid and Glioblastoma Cell Coculture on the Formation of Endothelial Cell Networks in Gelatin Hydrogels.Tissue Force Programs Cell Fate and Tumor Aggression.MiR-3662 suppresses hepatocellular carcinoma growth through inhibition of HIF-1α-mediated Warburg effect.Tissue and cellular rigidity and mechanosensitive signaling activation in Alexander disease.Phenotypic Basis for Matrix Stiffness-Dependent Chemoresistance of Breast Cancer Cells to Doxorubicin Glioblastoma Chemoresistance: The Double Play by Microenvironment and Blood-Brain Barrier

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Tissue mechanics promote IDH1-dependent HIF1α-tenascin C feedback to regulate glioblastoma aggression.

http://www.wikidata.org/entity/Q38732546

description

2016 nî lūn-bûn

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name

Tissue mechanics promote IDH1- ...... ulate glioblastoma aggression.

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type

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Tissue mechanics promote IDH1- ...... ulate glioblastoma aggression.

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Tissue mechanics promote IDH1- ...... ulate glioblastoma aggression.

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Nature Cell Biology

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Tissue mechanics promote IDH1- ...... ulate glioblastoma aggression.

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Eric C Holland

http://www.w3.org/2001/XMLSchema#string

Gerald F Reis

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J Matthew Barnes

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Janna K Mouw

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Joanna J Phillips

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Johnathan N Lakins

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Khadjia Lobo

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Michael W Pickup

http://www.w3.org/2001/XMLSchema#string

Tracy R McKnight

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Yekaterina A Miroshnikova

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VEGF inhibits tumor cell invasion and mesenchymal transition through a MET/VEGFR2 complex

Identification of a CpG island methylator phenotype that defines a distinct subgroup of glioma

Oncometabolite 2-hydroxyglutarate is a competitive inhibitor of α-ketoglutarate-dependent dioxygenases

Matrix crosslinking forces tumor progression by enhancing integrin signaling

IDH1 and IDH2 mutations in gliomas

Comprehensive genomic characterization defines human glioblastoma genes and core pathways

Comprehensive, Integrative Genomic Analysis of Diffuse Lower-Grade Gliomas.

Tensional homeostasis and the malignant phenotype

MicroRNA-203 Modulates the Radiation Sensitivity of Human Malignant Glioma Cells

The somatic genomic landscape of glioblastoma

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1336-1345

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scholarly article

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10.1038/NCB3429

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12

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18

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Anders I Persson

Valerie M Weaver

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2016-11-07T00:00:00Z

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1033729399

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27820599

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5361403

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