Functional assessment of SLC4A11, an integral membrane protein mutated in corneal dystrophies.
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SLC4A11 Three-Dimensional Homology Model Rationalizes Corneal Dystrophy-Causing Mutations.Conditionally Immortal Slc4a11-/- Mouse Corneal Endothelial Cell Line Recapitulates Disrupted Glutaminolysis Seen in Slc4a11-/- Mouse Model.SLC4A11 depletion impairs NRF2 mediated antioxidant signaling and increases reactive oxygen species in human corneal endothelial cells during oxidative stressVps35-deficiency impairs SLC4A11 trafficking and promotes corneal dystrophy.Mouse Slc4a11 expressed in Xenopus oocytes is an ideally selective H+/OH- conductance pathway that is stimulated by rises in intracellular and extracellular pH.H(OH), H(OH), H(OH): a holiday perspective. Focus on "Mouse Slc4a11 expressed in Xenopus oocytes is an ideally selective H+/OH- conductance pathway that is stimulated by rises in intracellular and extracellular pH".
P2860
Functional assessment of SLC4A11, an integral membrane protein mutated in corneal dystrophies.
description
2016 nî lūn-bûn
@nan
2016年の論文
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2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
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name
Functional assessment of SLC4A ...... utated in corneal dystrophies.
@en
type
label
Functional assessment of SLC4A ...... utated in corneal dystrophies.
@en
prefLabel
Functional assessment of SLC4A ...... utated in corneal dystrophies.
@en
P2093
P2860
P1476
Functional assessment of SLC4A ...... utated in corneal dystrophies.
@en
P2093
Hans-Peter Schneider
Joachim W Deitmer
Sampath K Loganathan
P2860
P304
P356
10.1152/AJPCELL.00078.2016
P577
2016-08-24T00:00:00Z