Glucocorticoid Receptor ChIP-Seq Identifies PLCD1 as a KLF15 Target that Represses Airway Smooth Muscle Hypertrophy.

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[EXPRESS] Intrathecal administration of AYX2 DNA-decoy produces a long-term pain treatment in rat models of chronic pain by inhibiting the KLF6, KLF9 and KLF15 transcription factors.The -Omic Approach to Understanding Glucocorticoid Effects in Smooth Muscle: Diving for Pearls.

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Glucocorticoid Receptor ChIP-Seq Identifies PLCD1 as a KLF15 Target that Represses Airway Smooth Muscle Hypertrophy.

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Glucocorticoid Receptor ChIP-S ...... way Smooth Muscle Hypertrophy.

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Glucocorticoid Receptor ChIP-S ...... way Smooth Muscle Hypertrophy.

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Glucocorticoid Receptor ChIP-S ...... way Smooth Muscle Hypertrophy.

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Glucocorticoid Receptor ChIP-S ...... way Smooth Muscle Hypertrophy.

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Anthony N Gerber

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Reynold A Panettieri

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Sarah K Sasse

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Tzu L Phang

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Vineela Kadiyala

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P2860

Kruppel-like factor 15 is a regulator of cardiomyocyte hypertrophy.

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Cyclin G1 has growth inhibitory activity linked to the ARF-Mdm2-p53 and pRb tumor suppressor pathways

Moderated estimation of fold change and dispersion for RNA-seq data with DESeq2

Foxo transcription factors induce the atrophy-related ubiquitin ligase atrogin-1 and cause skeletal muscle atrophy

RNA-Seq transcriptome profiling identifies CRISPLD2 as a glucocorticoid responsive gene that modulates cytokine function in airway smooth muscle cells.

The adaptor protein insulin receptor substrate 2 inhibits alternative macrophage activation and allergic lung inflammation

Glucocorticoid receptor binds half sites as a monomer and regulates specific target genes

Glucocorticoid-induced changes in gene expression of airway smooth muscle in patients with asthma.

Pro-asthmatic cytokines regulate unliganded and ligand-dependent glucocorticoid receptor signaling in airway smooth muscle.

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226-237

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scholarly article

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10.1165/RCMB.2016-0357OC

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Thomas Danhorn

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2017-04-04T00:00:00Z

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Glucocorticoid Receptor ChIP-Seq Identifies PLCD1 as a KLF15 Target that Represses Airway Smooth Muscle Hypertrophy.

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Glucocorticoid Receptor ChIP-Seq Identifies PLCD1 as a KLF15 Target that Represses Airway Smooth Muscle Hypertrophy.

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