Hypercontractile mutant of ventricular myosin essential light chain leads to disruption of sarcomeric structure and function and results in restrictive cardiomyopathy in mice.

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Sarcomeric perturbations of myosin motors lead to dilated cardiomyopathy in genetically modified MYL2 mice.Single cardiac ventricular myosins are autonomous motors.

P2860

Q49790570-A48DD81A-8D77-4B96-8CE1-823895FC42BF Q52579753-D2DA95C5-5BAC-4268-84B1-04541AAB913B

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Hypercontractile mutant of ventricular myosin essential light chain leads to disruption of sarcomeric structure and function and results in restrictive cardiomyopathy in mice.

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http://www.wikidata.org/entity/Q38735729

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2017 nî lūn-bûn

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2017年の論文

@ja

2017年論文

@yue

2017年論文

@zh-hant

2017年論文

@zh-hk

2017年論文

@zh-mo

2017年論文

@zh-tw

2017年论文

@wuu

2017年论文

@zh

2017年论文

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Hypercontractile mutant of ven ...... ictive cardiomyopathy in mice.

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label

Hypercontractile mutant of ven ...... ictive cardiomyopathy in mice.

@en

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Hypercontractile mutant of ven ...... ictive cardiomyopathy in mice.

@en

P1476

Hypercontractile mutant of ven ...... ictive cardiomyopathy in mice.

@en

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Aldrin V Gomes

http://www.w3.org/2001/XMLSchema#string

Chen-Ching Yuan

http://www.w3.org/2001/XMLSchema#string

Danuta Szczesna-Cordary

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Jingsheng Liang

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Katarzyna Kazmierczak

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Rosemeire Kanashiro-Takeuchi

http://www.w3.org/2001/XMLSchema#string

Thomas C Irving

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Thomas P Burghardt

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Yihua Wang

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P2860

Mutations in either the essential or regulatory light chains of myosin are associated with a rare myopathy in human heart and skeletal muscle

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Myosin light chain mutation causes autosomal recessive cardiomyopathy with mid-cavitary hypertrophy and restrictive physiology

Idiopathic restrictive cardiomyopathy in children is caused by mutations in cardiac sarcomere protein genes

Cardiac myosin light chain kinase is necessary for myosin regulatory light chain phosphorylation and cardiac performance in vivo

Contemporary definitions and classification of the cardiomyopathies: an American Heart Association Scientific Statement from the Council on Clinical Cardiology, Heart Failure and Transplantation Committee; Quality of Care and Outcomes Research and F

Mechanistic Pathways of Sex Differences in Cardiovascular Disease.

The Qdot-labeled actin super-resolution motility assay measures low-duty cycle muscle myosin step size.

N-Terminus of Cardiac Myosin Essential Light Chain Modulates Myosin Step-Size.

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scholarly article

P356

10.1093/CVR/CVX060

http://www.w3.org/2001/XMLSchema#string

P577

2017-03-23T00:00:00Z

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P698

28371863

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P932

5852631

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Hypercontractile mutant of ventricular myosin essential light chain leads to disruption of sarcomeric structure and function and results in restrictive cardiomyopathy in mice.

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P2860

P2860

Hypercontractile mutant of ventricular myosin essential light chain leads to disruption of sarcomeric structure and function and results in restrictive cardiomyopathy in mice.

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P1433

P1476

P2093

P2860

P31

P356

P577

P698

P932

P356

P1433

P1476

P2093

P2860

P31

P356

P577

P698

P932