Impairment of DYRK2 augments stem-like traits by promoting KLF4 expression in breast cancer.

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Downregulation of dual-specificity tyrosine-regulated kinase 2 promotes tumor cell proliferation and invasion by enhancing cyclin-dependent kinase 14 expression in breast cancer.

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Q48200216-F29A852E-A8BA-4BC7-A050-8C58A1D7CBCF

P2860

Impairment of DYRK2 augments stem-like traits by promoting KLF4 expression in breast cancer.

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2016 nî lūn-bûn

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2016年の論文

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Impairment of DYRK2 augments s ...... 4 expression in breast cancer.

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Impairment of DYRK2 augments s ...... 4 expression in breast cancer.

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Impairment of DYRK2 augments s ...... 4 expression in breast cancer.

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Impairment of DYRK2 augments s ...... 4 expression in breast cancer.

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H Takeyama

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K Yoshida

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R Mimoto

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S Hirooka

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Y Imawari

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P2860

DYRK2 is targeted to the nucleus and controls p53 via Ser46 phosphorylation in the apoptotic response to DNA damage

Transcriptional profiling of Krüppel-like factor 4 reveals a function in cell cycle regulation and epithelial differentiation

The epithelial-mesenchymal transition generates cells with properties of stem cells

Prospective identification of tumorigenic breast cancer cells

The MAPPER database: a multi-genome catalog of putative transcription factor binding sites

Epithelial-mesenchymal transitions in development and disease

Induction of pluripotent stem cells from mouse embryonic and adult fibroblast cultures by defined factors

Molecular definition of breast tumor heterogeneity

TRANSFAC: an integrated system for gene expression regulation.

Generation of breast cancer stem cells through epithelial-mesenchymal transition

P2888

onc.2016.349

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1862-1872

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scholarly article

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10.1038/ONC.2016.349

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Impairment of DYRK2 augments stem-like traits by promoting KLF4 expression in breast cancer.

about

P2860

P2860

Impairment of DYRK2 augments stem-like traits by promoting KLF4 expression in breast cancer.

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