DRα1-MOG-35-55 treatment reduces lesion volumes and improves neurological deficits after traumatic brain injury.

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Huperzine A alleviates neuroinflammation, oxidative stress and improves cognitive function after repetitive traumatic brain injury.Potential immunotherapies for traumatic brain and spinal cord injury.

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DRα1-MOG-35-55 treatment reduces lesion volumes and improves neurological deficits after traumatic brain injury.

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http://www.wikidata.org/entity/Q38743804

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年论文

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2017年论文

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2017年论文

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name

DRα1-MOG-35-55 treatment reduc ...... after traumatic brain injury.

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type

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DRα1-MOG-35-55 treatment reduc ...... after traumatic brain injury.

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DRα1-MOG-35-55 treatment reduc ...... after traumatic brain injury.

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DRα1-MOG-35-55 treatment reduc ...... after traumatic brain injury.

@en

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Arthur A Vandenbark

http://www.w3.org/2001/XMLSchema#string

Gil Benedek

http://www.w3.org/2001/XMLSchema#string

Halina Offner

http://www.w3.org/2001/XMLSchema#string

Honglei Ren

http://www.w3.org/2001/XMLSchema#string

Lei Zhang

http://www.w3.org/2001/XMLSchema#string

Li Ren

http://www.w3.org/2001/XMLSchema#string

Liu Yang

http://www.w3.org/2001/XMLSchema#string

Minshu Li

http://www.w3.org/2001/XMLSchema#string

Roberto Meza-Romero

http://www.w3.org/2001/XMLSchema#string

Siman Gao

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Quantitative map of multiple auditory cortical regions with a stereotaxic fine-scale atlas of the mouse brain

Inflammation and neuroprotection in traumatic brain injury

Emerging Roles for the Immune System in Traumatic Brain Injury

Subacute intranasal administration of tissue plasminogen activator promotes neuroplasticity and improves functional recovery following traumatic brain injury in rats

HLA-DRα1 constructs block CD74 expression and MIF effects in experimental autoimmune encephalomyelitis.

Novel humanized recombinant T cell receptor ligands protect the female brain after experimental stroke

Recombinant T cell receptor ligand treatment improves neurological outcome in the presence of tissue plasminogen activator in experimental ischemic stroke.

Traumatic brain injury causes selective, CD74-dependent peripheral lymphocyte activation that exacerbates neurodegeneration.

Preclinical evaluation of recombinant T cell receptor ligand RTL1000 as a therapeutic agent in ischemic stroke

Therapy with recombinant T-cell receptor ligand reduces infarct size and infiltrating inflammatory cells in brain after middle cerebral artery occlusion in mice

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s11011-017-9991-6

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scholarly article

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10.1007/S11011-017-9991-6

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2017-03-16T00:00:00Z

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1084028731

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28303450

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traumatic brain injury

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5600636

http://www.w3.org/2001/XMLSchema#string

DRα1-MOG-35-55 treatment reduces lesion volumes and improves neurological deficits after traumatic brain injury.

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P2860

DRα1-MOG-35-55 treatment reduces lesion volumes and improves neurological deficits after traumatic brain injury.

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