MEK and TAK1 Regulate Apoptosis in Colon Cancer Cells with KRAS-Dependent Activation of Proinflammatory Signaling.

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MEK and TAK1 Regulate Apoptosis in Colon Cancer Cells with KRAS-Dependent Activation of Proinflammatory Signaling.

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2016 nî lūn-bûn

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2016年の論文

@ja

2016年論文

@yue

2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

@zh-tw

2016年论文

@wuu

2016年论文

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2016年论文

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MEK and TAK1 Regulate Apoptosi ...... of Proinflammatory Signaling.

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Item

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MEK and TAK1 Regulate Apoptosi ...... of Proinflammatory Signaling.

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MEK and TAK1 Regulate Apoptosi ...... of Proinflammatory Signaling.

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P1476

MEK and TAK1 Regulate Apoptosi ...... of Proinflammatory Signaling.

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P2093

Anurag Singh

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Connor Kenny

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Kelsey L McNew

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Leah Ray

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Trevor J Grant

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William J Whipple

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P2860

Disruption of CRAF-mediated MEK activation is required for effective MEK inhibition in KRAS mutant tumors

Synthetic lethal interaction of combined BCL-XL and MEK inhibition promotes tumor regressions in KRAS mutant cancer models

mTOR: from growth signal integration to cancer, diabetes and ageing

A genome-wide RNAi screen identifies multiple synthetic lethal interactions with the Ras oncogene

Inflammation and cancer

Co-occurring genomic alterations define major subsets of KRAS-mutant lung adenocarcinoma with distinct biology, immune profiles, and therapeutic vulnerabilities.

Unresponsiveness of colon cancer to BRAF(V600E) inhibition through feedback activation of EGFR.

The GATA2 transcriptional network is requisite for RAS oncogene-driven non-small cell lung cancer.

A genetic model for colorectal tumorigenesis

Pattern recognition receptors and inflammation

P304

1204-1216

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P31

scholarly article

P356

10.1158/1541-7786.MCR-16-0173

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P433

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P478

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P50

Anita Kuldeep Mehta

P577

2016-09-21T00:00:00Z

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P698

27655129

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P932

5136310

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