Mitochondrial Neurogastrointestinal Encephalomyopathy Caused by Thymidine Phosphorylase Enzyme Deficiency: From Pathogenesis to Emerging Therapeutic Options.
about
Hemodialysis in MNGIE transiently reduces serum and urine levels of thymidine and deoxyuridine, but not CSF levels and neurological function.Nutritional Interventions for Mitochondrial OXPHOS Deficiencies: Mechanisms and Model Systems.The clinical eye.Preclinical Efficacy and Safety Evaluation of Hematopoietic Stem Cell Gene Therapy in a Mouse Model of MNGIE.Transplantation, gene therapy and intestinal pathology in MNGIE patients and miceMitochondrial Disease: Advances in clinical diagnosis, management, therapeutic development, and preventative strategies
P2860
Mitochondrial Neurogastrointestinal Encephalomyopathy Caused by Thymidine Phosphorylase Enzyme Deficiency: From Pathogenesis to Emerging Therapeutic Options.
description
2017 nî lūn-bûn
@nan
2017年の論文
@ja
2017年論文
@yue
2017年論文
@zh-hant
2017年論文
@zh-hk
2017年論文
@zh-mo
2017年論文
@zh-tw
2017年论文
@wuu
2017年论文
@zh
2017年论文
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name
Mitochondrial Neurogastrointes ...... Emerging Therapeutic Options.
@en
type
label
Mitochondrial Neurogastrointes ...... Emerging Therapeutic Options.
@en
prefLabel
Mitochondrial Neurogastrointes ...... Emerging Therapeutic Options.
@en
P2093
P2860
P356
P1476
Mitochondrial Neurogastrointes ...... Emerging Therapeutic Options.
@en
P2093
Irenaeus F M de Coo
Niek P van Til
Peter Sillevis Smitt
Rana Yadak
P2860
P356
10.3389/FNCEL.2017.00031
P577
2017-02-15T00:00:00Z