Rictor has a pivotal role in maintaining quiescence as well as stemness of leukemia stem cells in MLL-driven leukemia.

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Inhibition of mTOR kinase as a therapeutic target for acute myeloid leukemia.Decitabine-Induced Changes in Human Myelodysplastic Syndrome Cell Line SKM-1 Are Mediated by FOXO3A Activation.Biological Aspects of mTOR in Leukemia

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P2860

Rictor has a pivotal role in maintaining quiescence as well as stemness of leukemia stem cells in MLL-driven leukemia.

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P2860

Phosphorylation and regulation of Akt/PKB by the rictor-mTOR complex

FoxOs inhibit mTORC1 and activate Akt by inducing the expression of Sestrin3 and Rictor

mTOR signaling in growth control and disease

mTOR complex 2 is required for the development of prostate cancer induced by Pten loss in mice

Inhibition of BET recruitment to chromatin as an effective treatment for MLL-fusion leukaemia

RICTOR Amplification Defines a Novel Subset of Patients with Lung Cancer Who May Benefit from Treatment with mTORC1/2 Inhibitors

Rictor/mTORC2 is essential for maintaining a balance between beta-cell proliferation and cell size

Pten dependence distinguishes haematopoietic stem cells from leukaemia-initiating cells

mTOR and cancer: insights into a complex relationship

Selective killing of mixed lineage leukemia cells by a potent small-molecule DOT1L inhibitor.

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leu.2016.223

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10.1038/LEU.2016.223

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Rictor has a pivotal role in maintaining quiescence as well as stemness of leukemia stem cells in MLL-driven leukemia.

about

P2860

P2860

Rictor has a pivotal role in maintaining quiescence as well as stemness of leukemia stem cells in MLL-driven leukemia.

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