PARP inhibition ameliorates nephropathy in an animal model of type 2 diabetes: focus on oxidative stress, inflammation, and fibrosis.

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Actinidia chinensis Planch. Improves the Indices of Antioxidant and Anti-Inflammation Status of Type 2 Diabetes Mellitus by Activating Keap1 and Nrf2 via the Upregulation of MicroRNA-424.

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PARP inhibition ameliorates nephropathy in an animal model of type 2 diabetes: focus on oxidative stress, inflammation, and fibrosis.

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2017年論文

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PARP inhibition ameliorates ne ...... s, inflammation, and fibrosis.

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PARP inhibition ameliorates ne ...... s, inflammation, and fibrosis.

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PARP inhibition ameliorates ne ...... s, inflammation, and fibrosis.

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P1476

PARP inhibition ameliorates ne ...... ss, inflammation, and fibrosis

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Ahmed F Ahmed

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Esraa M Zakaria

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Hany M El-Bassossy

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Nabila N El-Maraghy

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P2860

A physical and functional map of the human TNF-alpha/NF-kappa B signal transduction pathway

Biochemistry and molecular cell biology of diabetic complications

Molecular mechanisms of diabetic renal hypertrophy.

Functions of poly(ADP-ribose) polymerase (PARP) in DNA repair, genomic integrity and cell death.

Poly(ADP-ribose) polymerase-1 dependence of stress-induced transcription factors and associated gene expression in glia.

Poly(ADP-ribose) polymerase-1 (PARP-1) gene deficiency alleviates diabetic kidney disease.

Poly(ADP-ribose) polymerase (PARP) inhibition counteracts multiple manifestations of kidney disease in long-term streptozotocin-diabetic rat model

Abroma augusta L. (Malvaceae) leaf extract attenuates diabetes induced nephropathy and cardiomyopathy via inhibition of oxidative stress and inflammatory response.

Xanthine oxidase inhibition alleviates the cardiac complications of insulin resistance: effect on low grade inflammation and the angiotensin system.

Inhibition of the activity of poly(ADP ribose) synthetase reduces ischemia-reperfusion injury in the heart and skeletal muscle.

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s00210-017-1360-9

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621-631

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scholarly article

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10.1007/S00210-017-1360-9

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1083855045

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PARP inhibition ameliorates nephropathy in an animal model of type 2 diabetes: focus on oxidative stress, inflammation, and fibrosis.

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P2860

P2860

PARP inhibition ameliorates nephropathy in an animal model of type 2 diabetes: focus on oxidative stress, inflammation, and fibrosis.

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