NO-independent activation of soluble guanylate cyclase prevents disease progression in rats with 5/6 nephrectomy.
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NO-independent stimulators and activators of soluble guanylate cyclase: discovery and therapeutic potentialBreakthrough in heart failure with preserved ejection fraction: are we there yet?Cardiac endothelium-myocyte interaction: clinical opportunities for new heart failure therapies regardless of ejection fractionThe cGMP signaling pathway as a therapeutic target in heart failure with preserved ejection fractionEffects of DPP-4 inhibitors on the heart in a rat model of uremic cardiomyopathyChronic Activation of Heme Free Guanylate Cyclase Leads to Renal Protection in Dahl Salt-Sensitive Rats.Development of Therapeutics That Induce Mitochondrial Biogenesis for the Treatment of Acute and Chronic Degenerative Diseases.Cyclic nucleotide signalling in kidney fibrosis.Arteriolar function in visceral adipose tissue is impaired in human obesityThe soluble guanylyl cyclase activator bay 58-2667 selectively limits cardiomyocyte hypertrophy.Cinaciguat prevents the development of pathologic hypertrophy in a rat model of left ventricular pressure overload.Experimental models of renal disease and the cardiovascular system.Soluble guanylate cyclase: a potential therapeutic target for heart failure.Redox signaling as a therapeutic target to inhibit myofibroblast activation in degenerative fibrotic diseaseSoluble Guanylate Cyclase Stimulators: a Novel Treatment Option for Heart Failure Associated with Cardiorenal Syndromes?Antioxidant treatment reverts increased arterial Basal tone and oxidative stress in nephrectomized (5/6) hypertensive rats.The adenosine A1 receptor antagonist SLV320 reduces myocardial fibrosis in rats with 5/6 nephrectomy without affecting blood pressure.Cinaciguat ameliorates glomerular damage by reducing ERK1/2 activity and TGF-ß expression in type-1 diabetic ratsChronic intratracheal application of the soluble guanylyl cyclase stimulator BAY 41-8543 ameliorates experimental pulmonary hypertension.Increased TG2 expression can result in induction of transforming growth factor beta1, causing increased synthesis and deposition of matrix proteins, which can be regulated by nitric oxide.Cytochrome b5 Reductase 3 Modulates Soluble Guanylate Cyclase Redox State and cGMP Signaling.Pressure-independent effects of pharmacological stimulation of soluble guanylate cyclase on fibrosis in pressure-overloaded rat heart.Role of soluble guanylate cyclase in renal hemodynamics and autoregulation in the rat.Redox control of vascular smooth muscle cell function and plasticity.A Soluble Guanylate Cyclase Activator Inhibits the Progression of Diabetic Nephropathy in the ZSF1 Rat.
P2860
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P2860
NO-independent activation of soluble guanylate cyclase prevents disease progression in rats with 5/6 nephrectomy.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
NO-independent activation of s ...... in rats with 5/6 nephrectomy.
@en
type
label
NO-independent activation of s ...... in rats with 5/6 nephrectomy.
@en
prefLabel
NO-independent activation of s ...... in rats with 5/6 nephrectomy.
@en
P2093
P2860
P356
P1476
NO-independent activation of s ...... in rats with 5/6 nephrectomy.
@en
P2093
Andreas Hucke
Christiane Rothkegel
Katharina Relle
Michael Godes
Philipp Kalk
P2860
P304
P356
10.1038/SJ.BJP.0706792
P407
P577
2006-06-12T00:00:00Z