Deoxynivalenol induced mouse skin tumor initiation: Elucidation of molecular mechanisms in human HaCaT keratinocytes.

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Risks to human and animal health related to the presence of deoxynivalenol and its acetylated and modified forms in food and feed

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Deoxynivalenol induced mouse skin tumor initiation: Elucidation of molecular mechanisms in human HaCaT keratinocytes.

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http://www.wikidata.org/entity/Q38759236

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2016 nî lūn-bûn

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

@zh-hk

2016年論文

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2016年論文

@zh-tw

2016年论文

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2016年论文

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2016年论文

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name

Deoxynivalenol induced mouse s ...... in human HaCaT keratinocytes.

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type

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Deoxynivalenol induced mouse s ...... in human HaCaT keratinocytes.

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Deoxynivalenol induced mouse s ...... in human HaCaT keratinocytes.

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P1476

Deoxynivalenol induced mouse s ...... in human HaCaT keratinocytes.

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Bhushan P Chaudhari

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Haushila P Pandey

http://www.w3.org/2001/XMLSchema#string

Mukul Das

http://www.w3.org/2001/XMLSchema#string

Prachi Tewari

http://www.w3.org/2001/XMLSchema#string

Premendra D Dwivedi

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P2860

Rapid colorimetric assay for cellular growth and survival: Application to proliferation and cytotoxicity assays

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Cell survival responses to environmental stresses via the Keap1-Nrf2-ARE pathway

Studies on the mechanism of skin tumor promotion: evidence for several stages in promotion.

Diverse functions of JNK signaling and c-Jun in stress response and apoptosis.

The contribution of endogenous sources of DNA damage to the multiple mutations in cancer.

Topical application of ochratoxin A causes DNA damage and tumor initiation in mouse skin.

ROS-generating mitochondrial DNA mutations can regulate tumor cell metastasis.

JNK signaling in apoptosis

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2033-2046

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scholarly article

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10.1002/IJC.30260

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139

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Sakshi Mishra

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2016-07-08T00:00:00Z

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27389473

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P921

deoxynivalenol

Deoxynivalenol induced mouse skin tumor initiation: Elucidation of molecular mechanisms in human HaCaT keratinocytes.

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P2860

P2860

Deoxynivalenol induced mouse skin tumor initiation: Elucidation of molecular mechanisms in human HaCaT keratinocytes.

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