Mathematical Modeling of E6-p53 interactions in Cervical Cancer

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Mathematical Modeling of E6-p53 interactions in Cervical Cancer

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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Mathematical Modeling of E6-p53 interactions in Cervical Cancer

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Mathematical Modeling of E6-p53 interactions in Cervical Cancer

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Mathematical Modeling of E6-p53 interactions in Cervical Cancer

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P1476

Mathematical Modeling of E6-p53 interactions in Cervical Cancer

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P2093

A I Bhatti

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Faryal Khattak

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Muhammad Haseeb

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Mukhtar Ullah

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Sahar Fazal

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P2860

The cigarette smoke carcinogen benzo[a]pyrene enhances human papillomavirus synthesis

p53 mutations in human cancers

An in silico model of the ubiquitin-proteasome system that incorporates normal homeostasis and age-related decline.

HPV E6, E6AP and cervical cancer.

The role of the E6-p53 interaction in the molecular pathogenesis of HPV.

Genus beta human papillomavirus E6 proteins vary in their effects on the transactivation of p53 target genes.

Cervical carcinoma in a Muslim community.

The role of TP53 in Cervical carcinogenesis.

Chapter 3: Cofactors in human papillomavirus carcinogenesis--role of parity, oral contraceptives, and tobacco smoking.

Knock down of p53 or its ubiquitin ligase E6AP does not affect the sensitivity of human papillomavirus-positive cervical cancer cells to cisplatin.

P304

1057-1061

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P31

scholarly article

P356

10.22034/APJCP.2017.18.4.1057

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P433

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P478

http://www.w3.org/2001/XMLSchema#string

P577

2017-04-01T00:00:00Z

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P698

28547941

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P932

5494216

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