Deregulated E2F5/p38/SMAD3 Circuitry Reinforces the Pro-Tumorigenic Switch of TGFβ Signaling in Prostate Cancer.

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MicroRNA-1179 inhibits glioblastoma cell proliferation and cell cycle progression via directly targeting E2F transcription factor 5.Pathway-based expression profiling of benign prostatic hyperplasia and prostate cancer delineates an immunophilin molecule associated with cancer progression.

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Deregulated E2F5/p38/SMAD3 Circuitry Reinforces the Pro-Tumorigenic Switch of TGFβ Signaling in Prostate Cancer.

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2016 nî lūn-bûn

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Deregulated E2F5/p38/SMAD3 Cir ...... Signaling in Prostate Cancer.

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Deregulated E2F5/p38/SMAD3 Cir ...... Signaling in Prostate Cancer.

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Deregulated E2F5/p38/SMAD3 Cir ...... Signaling in Prostate Cancer.

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Deregulated E2F5/p38/SMAD3 Cir ...... Signaling in Prostate Cancer.

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Ankur Bhowal

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Pritha Mukherjee

http://www.w3.org/2001/XMLSchema#string

Sanghamitra Sengupta

http://www.w3.org/2001/XMLSchema#string

Sanmitra Basu

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Subhadipa Majumder

http://www.w3.org/2001/XMLSchema#string

Urmi Chatterji

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P2860

East meets West: ethnic differences in prostate cancer epidemiology between East Asians and Caucasians

Molecular mechanisms of epithelial-mesenchymal transition

Cancer statistics, 2013

TGFbeta signaling in growth control, cancer, and heritable disorders

TGFβ signalling in context

TGFbeta in Cancer

Comparative analysis of TGF-β/Smad signaling dependent cytostasis in human hepatocellular carcinoma cell lines.

E2F5 status significantly improves malignancy diagnosis of epithelial ovarian cancer

E2f1-3 switch from activators in progenitor cells to repressors in differentiating cells.

Two faces of TGF-beta1 in breast cancer.

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2482-2492

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scholarly article

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10.1002/JCP.25361

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231

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2016-02-25T00:00:00Z

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26919443

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Deregulated E2F5/p38/SMAD3 Circuitry Reinforces the Pro-Tumorigenic Switch of TGFβ Signaling in Prostate Cancer.

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P2860

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Deregulated E2F5/p38/SMAD3 Circuitry Reinforces the Pro-Tumorigenic Switch of TGFβ Signaling in Prostate Cancer.

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