UDP-sugar substrates of HAS3 regulate its O-GlcNAcylation, intracellular traffic, extracellular shedding and correlate with melanoma progression.
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Hijacking the Hexosamine Biosynthetic Pathway to Promote EMT-Mediated Neoplastic PhenotypesHuman Keratinocytes Respond to Extracellular UTP by Induction of Hyaluronan Synthase 2 Expression and Increased Hyaluronan Synthesis.High extent of O-GlcNAcylation in breast cancer cells correlates with the levels of HAS enzymes, accumulation of hyaluronan, and poor outcome.
P2860
UDP-sugar substrates of HAS3 regulate its O-GlcNAcylation, intracellular traffic, extracellular shedding and correlate with melanoma progression.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
UDP-sugar substrates of HAS3 r ...... ate with melanoma progression.
@en
type
label
UDP-sugar substrates of HAS3 r ...... ate with melanoma progression.
@en
prefLabel
UDP-sugar substrates of HAS3 r ...... ate with melanoma progression.
@en
P2093
P2860
P1476
UDP-sugar substrates of HAS3 r ...... ate with melanoma progression.
@en
P2093
Antti Hassinen
Ashik Jawahar Deen
Markku Tammi
Piia Takabe
Raija Tammi
Riikka Kärnä
Sakari Kellokumpu
Sanna Oikari
Sanna Pasonen-Seppänen
Sara Wojciechowski
P2860
P2888
P304
P356
10.1007/S00018-016-2158-5
P50
P577
2016-02-16T00:00:00Z