Necroptosis-independent signaling by the RIP kinases in inflammation.
about
The complex cascade of cellular events governing inflammasome activation and IL-1β processing in response to inhaled particlesRIPK3 Restricts Viral Pathogenesis via Cell Death-Independent Neuroinflammation.Complex Pathologic Roles of RIPK1 and RIPK3: Moving Beyond Necroptosis.RIPped for neuroinflammation.Necroptosis in microglia contributes to neuroinflammation and retinal degeneration through TLR4 activation.Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018.Endoplasmic reticulum chaperone GRP78 mediates cigarette smoke-induced necroptosis and injury in bronchial epithelium.The Receptor Interacting Protein Kinases in the Liver.Caspase-8 deficiency in mouse embryos triggers chronic RIPK1-dependent activation of inflammatory genes, independently of RIPK3.The kinase receptor-interacting protein 1 is required for inflammasome activation induced by endoplasmic reticulum stress.
P2860
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P2860
Necroptosis-independent signaling by the RIP kinases in inflammation.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Necroptosis-independent signaling by the RIP kinases in inflammation.
@en
type
label
Necroptosis-independent signaling by the RIP kinases in inflammation.
@en
prefLabel
Necroptosis-independent signaling by the RIP kinases in inflammation.
@en
P2860
P1476
Necroptosis-independent signaling by the RIP kinases in inflammation.
@en
P2093
Francis Ka-Ming Chan
Kenta Moriwaki
P2860
P2888
P304
P356
10.1007/S00018-016-2203-4
P577
2016-04-05T00:00:00Z