about
Autistic-like phenotypes in Cadps2-knockout mice and aberrant CADPS2 splicing in autistic patients.Animal models of autism spectrum disorder (ASD): a synaptic-level approach to autistic-like behavior in micePhospholipase D family member 4, a transmembrane glycoprotein with no phospholipase D activity, expression in spleen and early postnatal microgliaThe secretory granule-associated protein CAPS2 regulates neurotrophin release and cell survivalCalcium-dependent activator protein for secretion 2 (CAPS2) promotes BDNF secretion and is critical for the development of GABAergic interneuron network.Axonal localization of Ca2+-dependent activator protein for secretion 2 is critical for subcellular locality of brain-derived neurotrophic factor and neurotrophin-3 release affecting proper development of postnatal mouse cerebellum.Alternative splicing variations in mouse CAPS2: differential expression and functional properties of splicing variants.Lack of stress responses to long-term effects of corticosterone in Caps2 knockout mice.Mammalian-Specific Central Myelin Protein Opalin Is Redundant for Normal Myelination: Structural and Behavioral AssessmentsThe brain-specific RasGEF very-KIND is required for normal dendritic growth in cerebellar granule cells and proper motor coordination.Reduced axonal localization of a Caps2 splice variant impairs axonal release of BDNF and causes autistic-like behavior in mice.CAPS1 stabilizes the state of readily releasable synaptic vesicles to fusion competence at CA3-CA1 synapses in adult hippocampusDevelopmentally regulated Ca2+-dependent activator protein for secretion 2 (CAPS2) is involved in BDNF secretion and is associated with autism susceptibility.Mouse models of mutations and variations in autism spectrum disorder-associated genes: mice expressing Caps2/Cadps2 copy number and alternative splicing variants.Ca(2+)-dependent activator protein for secretion 2 and autistic-like phenotypes.Systematizing and cloning of genes involved in the cerebellar cortex circuit development.Puralpha, a single-stranded DNA binding protein, suppresses the enhancer activity of cAMP response element (CRE).Interaction of Ca(2+)-dependent activator protein for secretion 1 (CAPS1) with septin family proteins in mouse brain.Interaction of calcium-dependent activator protein for secretion 1 (CAPS1) with the class II ADP-ribosylation factor small GTPases is required for dense-core vesicle trafficking in the trans-Golgi network.CAPS1 deficiency perturbs dense-core vesicle trafficking and Golgi structure and reduces presynaptic release probability in the mouse brain.Phase advance of the light-dark cycle perturbs diurnal rhythms of brain-derived neurotrophic factor and neurotrophin-3 protein levels, which reduces synaptophysin-positive presynaptic terminals in the cortex of juvenile ratsExpression of the IP3R1 promoter-driven nls-lacZ transgene in Purkinje cell parasagittal arrays of developing mouse cerebellum.Increase of calmodulin III gene expression by mu-opioid receptor stimulation in PC12 cells.Response to the letter by Eran et al.Autistic-like behavioral phenotypes in a mouse model with copy number variation of the CAPS2/CADPS2 gene.Differential distributions of the Ca2+ -dependent activator protein for secretion family proteins (CAPS2 and CAPS1) in the mouse brain.CAPS2 deficiency affects environmental enrichment-induced adult neurogenesis and differentiation/survival of newborn neurons in the hippocampal dentate gyrus.Impaired cerebellar development and function in mice lacking CAPS2, a protein involved in neurotrophin release.Tissue distribution of Ca2+-dependent activator protein for secretion family members CAPS1 and CAPS2 in mice.Analysis of gene expression in Ca2+-dependent activator protein for secretion 2 (Cadps2) knockout cerebellum using GeneChip and KEGG pathways.Age-dependent redistribution and hypersialylation of the central myelin paranodal loop membrane protein Opalin in the mouse brain.The effects of amiloride on the labellar taste receptor cells of the fleshfly Boettcherisca peregrina.Calcium-dependent activator protein for secretion 2 (CADPS2) deficiency causes abnormal synapse development in hippocampal mossy fiber terminalsAspects of excitatory/inhibitory synapses in multiple brain regions are correlated with levels of brain-derived neurotrophic factor/neurotrophin-3Deletion of Class II ADP-Ribosylation Factors in Mice Causes Tremor by the Nav1.6 Loss in Cerebellar Purkinje Cell Axon Initial Segments
P50
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P50
description
hulumtues
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Tetsushi Sadakata
@ast
Tetsushi Sadakata
@en
Tetsushi Sadakata
@es
Tetsushi Sadakata
@nl
Tetsushi Sadakata
@sl
type
label
Tetsushi Sadakata
@ast
Tetsushi Sadakata
@en
Tetsushi Sadakata
@es
Tetsushi Sadakata
@nl
Tetsushi Sadakata
@sl
prefLabel
Tetsushi Sadakata
@ast
Tetsushi Sadakata
@en
Tetsushi Sadakata
@es
Tetsushi Sadakata
@nl
Tetsushi Sadakata
@sl
P106
P108
P21
P31
P496
0000-0003-0276-4162
P569
2000-01-01T00:00:00Z