Lipid-Induced Signaling Causes Release of Inflammatory Extracellular Vesicles From Hepatocytes. - Wikidata - wikimedia - TriplyDB

Lipid-Induced Signaling Causes Release of Inflammatory Extracellular Vesicles From Hepatocytes.

Lipid-Induced Signaling Causes Release of Inflammatory Extracellular Vesicles From Hepatocytes.

http://www.wikidata.org/entity/Q38803828

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Extracellular Vesicles: How the External and Internal Environment Can Shape Cell-To-Cell Communication.Hepatocyte-secreted extracellular vesicles modify blood metabolome and endothelial function by an arginase-dependent mechanism.High Dietary Fructose: Direct or Indirect Dangerous Factors Disturbing Tissue and Organ Functions Exosomes derived from palmitic acid-treated hepatocytes induce fibrotic activation of hepatic stellate cells Low levels of IgM antibodies recognizing oxidation-specific epitopes are associated with human non-alcoholic fatty liver disease.Microvesicles released from fat-laden cells promote activation of hepatocellular NLRP3 inflammasome: A pro-inflammatory link between lipotoxicity and non-alcoholic steatohepatitis.Subtoxic Alterations in Hepatocyte-Derived Exosomes: An Early Step in Drug-Induced Liver Injury?Functional Roles and Therapeutic Applications of Exosomes in Hepatocellular Carcinoma.Mitochondrial DNA-enriched microparticles promote acute-on-chronic alcoholic neutrophilia and hepatotoxicity Exosomes in liver pathology.Lipotoxic lethal and sublethal stress signaling in hepatocytes: relevance to NASH pathogenesis.Extracellular vesicles in liver pathobiology: Small particles with big impact.Inhibition of sphingosine 1-phosphate signaling ameliorates murine nonalcoholic steatohepatitis.Extracellular vesicles in liver diseases.Liver inflammation and fibrosis.Liver macrophages in tissue homeostasis and disease.Extracellular vesicles in liver disease and potential as biomarkers and therapeutic targets.The role of the secretin/secretin receptor axis in inflammatory cholangiocyte communication via extracellular vesicles.Metabolomic profiling for identification of metabolites and relevant pathways for taurine in hepatic stellate cells.Mixed-lineage kinase 3 pharmacological inhibition attenuates murine nonalcoholic steatohepatitis.Lack of fibroblast growth factor 21 accelerates metabolic liver injury characterized by steatohepatities in mice.Biliary tract instillation of a SMAC mimetic induces TRAIL-dependent acute sclerosing cholangitis-like injury in mice.Therapeutic opportunities for alcoholic steatohepatitis and nonalcoholic steatohepatitis: exploiting similarities and differences in pathogenesis.Fibrogenic Signaling Is Suppressed in Hepatic Stellate Cells through Targeting of Connective Tissue Growth Factor (CCN2) by Cellular or Exosomal MicroRNA-199a-5p.Chemotherapy-Induced Tissue Injury: An Insight into the Role of Extracellular Vesicles-Mediated Oxidative Stress Responses.Extracellular vesicles released by fibroblasts undergoing H-Ras induced senescence show changes in lipid profile.Pathways of production and delivery of hepatocyte exosomes.Ezetimibe ameliorates steatohepatitis via AMP activated protein kinase-TFEB-mediated activation of autophagy and NLRP3 inflammasome inhibition.Exosomes as new players in metabolic organ cross-talk.Hepatic stellate cell-derived PDGFRα-enriched extracellular vesicles promote liver fibrosis in mice through SHP2.Macrophages and lipid metabolism.Non-alcoholic steatohepatitis pathogenesis: sublethal hepatocyte injury as a driver of liver inflammation.Cholangiocyte-derived exosomal long noncoding RNA H19 promotes cholestatic liver injury in mouse and human.Role of exosome-associated microRNA in diagnostic and therapeutic applications to metabolic disorders.Extracellular vesicles as potential biomarkers for alcohol- and drug-induced liver injury and their therapeutic applications.TRAIL Deletion Prevents Liver, but Not Adipose Tissue, Inflammation during Murine Diet-Induced Obesity.Therapeutic effects of serum extracellular vesicles in liver fibrosis.To die or not to die: death signaling in nonalcoholic fatty liver disease.The contrasting roles of inflammasomes in cancer.Noncoding RNAs Carried by Extracellular Vesicles in Endocrine Diseases.

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Lipid-Induced Signaling Causes Release of Inflammatory Extracellular Vesicles From Hepatocytes.

http://www.wikidata.org/entity/Q38803828

description

2016 nî lūn-bûn

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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2016年论文

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2016年论文

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name

Lipid-Induced Signaling Causes ...... lar Vesicles From Hepatocytes.

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Lipid-Induced Signaling Causes ...... lar Vesicles From Hepatocytes.

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Lipid-Induced Signaling Causes ...... lar Vesicles From Hepatocytes.

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J.GASTRO.2015.12.037

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Gastroenterology

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Lipid-Induced Signaling Causes ...... ular Vesicles From Hepatocytes

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Anuradha Krishnan

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Harmeet Malhi

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Michael R Charlton

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Nathan W Werneburg

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Samar H Ibrahim

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Steven F Bronk

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Vijay H Shah

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P2860

Rab27a and Rab27b control different steps of the exosome secretion pathway

Caspase-3-mediated cleavage of ROCK I induces MLC phosphorylation and apoptotic membrane blebbing

TRAIL activates JNK and NF-κB through RIP1-dependent and -independent pathways

Characterization of a functional NF-kappa B site in the human interleukin 1 beta promoter: evidence for a positive autoregulatory loop

Identification of RIP1 kinase as a specific cellular target of necrostatins

Extracellular vesicles: exosomes, microvesicles, and friends

Lipid-induced toxicity stimulates hepatocytes to release angiogenic microparticles that require Vanin-1 for uptake by endothelial cells.

Membrane blebbing during apoptosis results from caspase-mediated activation of ROCK I.

Exosome transfer from stromal to breast cancer cells regulates therapy resistance pathways

Death Receptor-Mediated Cell Death and Proinflammatory Signaling in Nonalcoholic Steatohepatitis.

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956-967

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scholarly article

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10.1053/J.GASTRO.2015.12.037

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4

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150

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Gregory J Gores

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2016-01-05T00:00:00Z

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290443364

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26764184

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