about
Expression and prognostic impact of the protein tyrosine phosphatases PRL-1, PRL-2, and PRL-3 in breast cancerVesicular transport: how many Ypt/Rab-GTPases make a eukaryotic cell?Targeting of syndecan-1 by micro-ribonucleic acid miR-10b modulates invasiveness of endometriotic cells via dysregulation of the proteolytic milieu and interleukin-6 secretionFunctions of cell surface heparan sulfate proteoglycansPhysicochemical and biological characterization of chitosan-microRNA nanocomplexes for gene delivery to MCF-7 breast cancer cellsEndometrial cells get side-tracked: side population cells promote epithelial-mesenchymal transition in endometrial carcinoma.The adult stem cell marker Musashi-1 modulates endometrial carcinoma cell cycle progression and apoptosis via Notch-1 and p21WAF1/CIP1.The full complement of yeast Ypt/Rab-GTPases and their involvement in exo- and endocytic trafficking.Syndecan-1 (CD138) modulates triple-negative breast cancer stem cell properties via regulation of LRP-6 and IL-6-mediated STAT3 signaling.microRNA miR-142-3p Inhibits Breast Cancer Cell Invasiveness by Synchronous Targeting of WASL, Integrin Alpha V, and Additional Cytoskeletal Elements.Syndecan-1 deficiency promotes tumor growth in a murine model of colitis-induced colon carcinomaHeparanase, hyaluronan, and CD44 in cancers: a breast carcinoma perspective.Constitutive and accelerated shedding of murine syndecan-1 is mediated by cleavage of its core protein at a specific juxtamembrane siteA Versatile Tool for Stable Inhibition of microRNA Activity.Syndecan-1 is a novel molecular marker for triple negative inflammatory breast cancer and modulates the cancer stem cell phenotype via the IL-6/STAT3, Notch and EGFR signaling pathwaysMicroRNAs in breast cancer pathogenesis.More than matrix: the multifaceted role of decorin in cancer.MicroRNA-dependent targeting of the extracellular matrix as a mechanism of regulating cell behavior.MicroRNA regulation of proteoglycan function in cancer.Differential roles for membrane-bound and soluble syndecan-1 (CD138) in breast cancer progression.Heparan sulphate as a regulator of leukocyte recruitment in inflammation.Syndecan-1 regulates dendritic cell migration in cutaneous hypersensitivity to haptens.Correlation between dioxin and endometriosis: an epigenetic route to unravel the pathogenesis of the disease.Estrogen receptor beta as epigenetic mediator of miR-10b and miR-145 in mammary cancer.MDA-MB-231 breast cancer cell viability, motility and matrix adhesion are regulated by a complex interplay of heparan sulfate, chondroitin-/dermatan sulfate and hyaluronan biosynthesis.Syndecan-4 expression is upregulated in endometriosis and contributes to an invasive phenotype.microRNA miR-200b affects proliferation, invasiveness and stemness of endometriotic cells by targeting ZEB1, ZEB2 and KLF4.Shed proteoglycans in tumor stroma.The anti-androgen drug dutasteride renders triple negative breast cancer cells more sensitive to chemotherapy via inhibition of HIF-1α-/VEGF-signaling.Influence of secreted frizzled receptor protein 1 (SFRP1) on neoadjuvant chemotherapy in triple negative breast cancer does not rely on WNT signaling.Roles and targeting of the HAS/hyaluronan/CD44 molecular system in cancer.miR-142-3p is a novel regulator of cell viability and proinflammatory signalling in endometrial stroma cells.Importance of transvaginal ultrasound applying elastography for identifying deep infiltrating endometriosis - a feasibility study.MicroRNA miR-145 inhibits proliferation, invasiveness, and stem cell phenotype of an in vitro endometriosis model by targeting multiple cytoskeletal elements and pluripotency factors.Syndecan-1 modulates β-integrin-dependent and interleukin-6-dependent functions in breast cancer cell adhesion, migration, and resistance to irradiation.Survivin, a target to modulate the radiosensitivity of Ewing's sarcoma.Targeting of syndecan-1 by microRNA miR-10b promotes breast cancer cell motility and invasiveness via a Rho-GTPase- and E-cadherin-dependent mechanism.Impact of testosterone on the expression of organic anion transporting polypeptides (OATP-1A2, OATP-2B1, OATP-3A1) in malignant and non-malignant human breast cells in vitro.Overlapping genes may control reprogramming of mouse somatic cells into induced pluripotent stem cells (iPSCs) and breast cancer stem cells.Targeting endothelin A receptor enhances anti-proliferative and anti-invasive effects of the HER2 antibody trastuzumab in HER2-overexpressing breast cancer cells.
P50
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P50
description
hulumtues
@sq
onderzoeker
@nl
researcher
@en
ricercatore
@it
հետազոտող
@hy
name
Martin Götte
@ast
Martin Götte
@en
Martin Götte
@es
Martin Götte
@sl
type
label
Martin Götte
@ast
Martin Götte
@en
Martin Götte
@es
Martin Götte
@sl
prefLabel
Martin Götte
@ast
Martin Götte
@en
Martin Götte
@es
Martin Götte
@sl
P108
P1053
G-2254-2018
P106
P1153
17634295500
P21
P213
0000 0000 1602 5932
P214
42145304957778610538
P31
P3829
P496
0000-0003-2360-2496
P734
P735
P7859
viaf-42145304957778610538