about
Cloning of novel human SEC14p-like proteins: ligand binding and functional propertiesTrophic factors in neurodegenerative disorders.Splicing changes in SMA mouse motoneurons and SMN-depleted neuroblastoma cells: evidence for involvement of splicing regulatory proteins.Central and peripheral defects in motor units of the diaphragm of spinal muscular atrophy mice.Mechanisms of axon degeneration: from development to disease.ER strikes again: Proteostasis Dysfunction In ALS.Selective neuronal vulnerability in neurodegenerative diseases: from stressor thresholds to degeneration.C9ORF72 Regulates Stress Granule Formation and Its Deficiency Impairs Stress Granule Assembly, Hypersensitizing Cells to Stress.Proteostasis impairment in ALS.Aberrant association of misfolded SOD1 with Na(+)/K(+)ATPase-α3 impairs its activity and contributes to motor neuron vulnerability in ALS.Marinesco-Sjögren syndrome protein SIL1 regulates motor neuron subtype-selective ER stress in ALS.Proteasomal inhibition alters the trafficking of the neurotrophin receptor TrkA.A role for motoneuron subtype-selective ER stress in disease manifestations of FALS mice.Differences in the surface binding and endocytosis of neurotrophins by p75NTR.Neuroprotection through excitability and mTOR required in ALS motoneurons to delay disease and extend survival.Impaired mTORC1-Dependent Expression of Homer-3 Influences SCA1 Pathophysiology.The small GTPase Rab7 controls the endosomal trafficking and neuritogenic signaling of the nerve growth factor receptor TrkA.Mitochondrial impairments contribute to Spinocerebellar ataxia type 1 progression and can be ameliorated by the mitochondria-targeted antioxidant MitoQ.Differential endocytic sorting of p75NTR and TrkA in response to NGF: a role for late endosomes in TrkA trafficking.Neurotrophin Receptors and Retrograde Signaling: A Long-Distance RelationshipHigh contrast staining for serial block face scanning electron microscopy without uranyl acetateEpidemiology of inherited cerebellar ataxias and challenges in clinical researchP2X1-regulated IL-22 secretion by innate lymphoid cells is required for efficient liver regenerationSelective vulnerability and pruning of phasic motoneuron axons in motoneuron disease alleviated by CNTFHuman genetics and neuropathology suggest a link between miR-218 and amyotrophic lateral sclerosis pathophysiology
P50
Q28205288-C7049E0F-0C5F-4ACD-A538-AEA2BE8DCB13Q35206104-FA2BFD74-3F8D-4E1C-8626-7E41C8921B47Q35563254-5A230DD8-D988-4B38-950B-97120C513BA5Q35856460-06727876-E27A-4B3B-ACF6-7DE2093A417DQ36932883-CF5F308B-24E2-4A72-8888-649FD412EDB4Q37149421-89786276-2F13-43E1-9F94-9EB3ADF1FDF9Q37899995-96A0A100-A755-49A7-9876-632E81F75452Q38781435-21803F11-1B45-483D-8215-57B5A55BAEF1Q38795183-648AEFA3-A751-4B74-AF41-646B8A0CD7D4Q41087321-C0EDD07C-BCEC-4FA1-BE30-957D2A585939Q42175672-0F5C7C2D-C20F-4F69-9B1C-2D2366FB5DE7Q45927835-DD979CF1-FB11-496F-960C-E08497BD7EFBQ46070173-474B2DC0-4B4A-40C4-BE58-7CE582445698Q47317636-9AFB4FAC-C619-406F-AD1C-ED0FE204A3D8Q48957960-06657736-D77B-4C05-BC0A-4D8A6092F520Q49020868-4230BDBF-FA6D-4A24-B11B-9F84279D5D7FQ50747092-D1947609-20F8-4289-AC89-4044EAA3AA22Q51653139-1FB3726C-C8B9-43E5-B958-5AAA9A72CA7DQ52562929-0430EB7F-7F23-4D2C-BA9D-88C14F2F88C6Q57374779-13647456-83A8-4F6E-8449-4D0C0D6FEDF3Q57979465-7D807DDC-1892-4127-9CAC-5317CC9AD956Q60220466-249CE9E5-6B40-4BCE-9EC9-8CB8DA113D1FQ60220469-D67146AA-A5E3-4D5D-8EFA-E42678B6460DQ60220472-105FE2DB-28E4-4B64-9AAC-C6E6327E4D8EQ92094258-BC8E4410-239A-4853-B99F-D5795B6C796E
P50
description
hulumtuese
@sq
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Smita Saxena
@ast
Smita Saxena
@en
Smita Saxena
@es
Smita Saxena
@sl
type
label
Smita Saxena
@ast
Smita Saxena
@en
Smita Saxena
@es
Smita Saxena
@sl
prefLabel
Smita Saxena
@ast
Smita Saxena
@en
Smita Saxena
@es
Smita Saxena
@sl
P106
P1153
8770170800
P21
P31
P496
0000-0003-4574-4591
P569
2000-01-01T00:00:00Z