Context-Specific Effects of TGF-β/SMAD3 in Cancer Are Modulated by the Epigenome. - Wikidata - wikimedia - TriplyDB

Context-Specific Effects of TGF-β/SMAD3 in Cancer Are Modulated by the Epigenome.

Context-Specific Effects of TGF-β/SMAD3 in Cancer Are Modulated by the Epigenome.

http://www.wikidata.org/entity/Q38810340

about

Patient-derived tumour xenografts for breast cancer drug discovery Smad3 Sensitizes Hepatocelluar Carcinoma Cells to Cisplatin by Repressing Phosphorylation of AKT.TGFβ promotes mesenchymal phenotype of pancreatic cancer cells, in part, through epigenetic activation of VAV1.TGF-β signaling in liver and gastrointestinal cancers Epigenetics and Cancer Stem Cells: Unleashing, Hijacking, and Restricting Cellular Plasticity.Regulation of the Cell Cycle and Inflammatory Arthritis by the Transcription Cofactor LBH Gene.The crossroads of breast cancer progression: insights into the modulation of major signaling pathways.Computational approach to discriminate human and mouse sequences in patient-derived tumour xenografts.Overexpression of LBH is associated with poor prognosis in human hepatocellular carcinoma.Transcriptomes define distinct subgroups of salivary gland adenoid cystic carcinoma with different driver mutations and outcomes.SOX4 can redirect TGF-β-mediated SMAD3-transcriptional output in a context-dependent manner to promote tumorigenesis

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Context-Specific Effects of TGF-β/SMAD3 in Cancer Are Modulated by the Epigenome.

http://www.wikidata.org/entity/Q38810340

description

2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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name

Context-Specific Effects of TGF-β/SMAD3 in Cancer Are Modulated by the Epigenome.

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Context-Specific Effects of TGF-β/SMAD3 in Cancer Are Modulated by the Epigenome.

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Context-Specific Effects of TGF-β/SMAD3 in Cancer Are Modulated by the Epigenome.

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Context-Specific Effects of TGF-β/SMAD3 in Cancer Are Modulated by the Epigenome

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Alejandra Bruna

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Ankita Sati Batra

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Oscar M Rueda

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Paul J Coffer

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Santiago Uribe-Lewis

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Stephin J Vervoort

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P2860

Model-based analysis of ChIP-Seq (MACS)

14-3-3ζ turns TGF-β's function from tumor suppressor to metastasis promoter in breast cancer by contextual changes of Smad partners from p53 to Gli2

Comprehensive molecular portraits of human breast tumours

The epithelial-mesenchymal transition generates cells with properties of stem cells

Fast and accurate short read alignment with Burrows-Wheeler transform

Smads bind directly to the Jun family of AP-1 transcription factors

Crystal structure of a Smad MH1 domain bound to DNA: insights on DNA binding in TGF-beta signaling

edgeR: a Bioconductor package for differential expression analysis of digital gene expression data

Integration of Smad and forkhead pathways in the control of neuroepithelial and glioblastoma cell proliferation

TGFβ signalling in context

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10.1016/J.CELREP.2015.11.040

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11

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Wendy Greenwood

Mae A Goldgraben

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287359853

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