Aspirin Inhibits LPS-Induced Expression of PI3K/Akt, ERK, NF-κB, CX3CL1, and MMPs in Human Bronchial Epithelial Cells.
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A novel co-drug of aspirin and ursolic acid interrupts adhesion, invasion and migration of cancer cells to vascular endothelium via regulating EMT and EGFR-mediated signaling pathways: multiple targets for cancer metastasis prevention and treatmentProteome-wide prediction of targets for aspirin: new insight into the molecular mechanism of aspirin.Aspirin-Mediated Acetylation Protects Against Multiple Neurodegenerative Pathologies by Impeding Protein Aggregation.Aspirin modulates the inflammatory response in a thrombus‑stimulated LMVEC model.
P2860
Aspirin Inhibits LPS-Induced Expression of PI3K/Akt, ERK, NF-κB, CX3CL1, and MMPs in Human Bronchial Epithelial Cells.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Aspirin Inhibits LPS-Induced E ...... an Bronchial Epithelial Cells.
@en
type
label
Aspirin Inhibits LPS-Induced E ...... an Bronchial Epithelial Cells.
@en
prefLabel
Aspirin Inhibits LPS-Induced E ...... an Bronchial Epithelial Cells.
@en
P2093
P2860
P1433
P1476
Aspirin Inhibits LPS-Induced E ...... an Bronchial Epithelial Cells.
@en
P2093
Jiannong Wu
Liling Wei
Lingcong Wang
Meifei Zhu
Ronglin Jiang
P2860
P2888
P304
P356
10.1007/S10753-015-0289-8
P577
2015-12-04T00:00:00Z
P6179
1008587360