Hyperaldosteronism: How to Discriminate Among Different Disease Forms? - Wikidata - wikimedia - TriplyDB

Hyperaldosteronism: How to Discriminate Among Different Disease Forms?

Hyperaldosteronism: How to Discriminate Among Different Disease Forms?

http://www.wikidata.org/entity/Q38823256

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Endocrine Tumors Causing Arterial Hypertension: Pathophysiological Mechanisms and Clinical Implications.

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Q39239777-7B85794E-216A-4717-9EC6-2730A471E43D

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Hyperaldosteronism: How to Discriminate Among Different Disease Forms?

http://www.wikidata.org/entity/Q38823256

description

2016 nî lūn-bûn

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

@zh-hk

2016年論文

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2016年論文

@zh-tw

2016年论文

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2016年论文

@zh

2016年论文

@zh-cn

name

Hyperaldosteronism: How to Discriminate Among Different Disease Forms?

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type

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Hyperaldosteronism: How to Discriminate Among Different Disease Forms?

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Hyperaldosteronism: How to Discriminate Among Different Disease Forms?

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S40292-016-0151-6

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High Blood Pressure & Cardiovascular Prevention

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Hyperaldosteronism: How to Discriminate Among Different Disease Forms?

@en

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Fabrizio Buffolo

http://www.w3.org/2001/XMLSchema#string

Franco Veglio

http://www.w3.org/2001/XMLSchema#string

Jacopo Burrello

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Martina Tetti

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Paolo Mulatero

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Valentina Crudo

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P2860

ARMC5 mutations in macronodular adrenal hyperplasia with Cushing's syndrome

Further evidence for linkage of familial hyperaldosteronism type II at chromosome 7p22 in Italian as well as Australian and South American families

Systematic review: diagnostic procedures to differentiate unilateral from bilateral adrenal abnormality in primary aldosteronism.

Somatic mutations in ATP1A1 and ATP2B3 lead to aldosterone-producing adenomas and secondary hypertension.

Somatic and germline CACNA1D calcium channel mutations in aldosterone-producing adenomas and primary aldosteronism

A case of severe hyperaldosteronism caused by a de novo mutation affecting a critical salt bridge Kir3.4 residue.

Understanding primary aldosteronism: impact of next generation sequencing and expression profiling.

Recurrent gain of function mutation in calcium channel CACNA1H causes early-onset hypertension with primary aldosteronism.

Primary Aldosteronism and ARMC5 Variants.

Immunohistochemical, genetic and clinical characterization of sporadic aldosterone-producing adenomas.

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s40292-016-0151-6

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203-208

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scholarly article

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10.1007/S40292-016-0151-6

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3

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23

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Silvia Monticone

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2016-05-02T00:00:00Z

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27136934

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