Advanced glycation end-product (AGE) induces apoptosis in human retinal ARPE-19 cells via promoting mitochondrial dysfunction and activating the Fas-FasL signaling.
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Novel nervous and multi-system regenerative therapeutic strategies for diabetes mellitus with mTORErythropoietin and mTOR: A "One-Two Punch" for Aging-Related Disorders Accompanied by Enhanced Life ExpectancyInhibition of Methylglyoxal-Induced AGEs/RAGE Expression Contributes to Dermal Protection by N-Acetyl-L-Cysteine.The bright side of reactive oxygen species: lifespan extension without cellular demise.Inhibition of Starvation-Triggered Endoplasmic Reticulum Stress, Autophagy, and Apoptosis in ARPE-19 Cells by Taurine through Modulating the Expression of Calpain-1 and Calpain-2.Long-term apoptosis-related protein expression in the diabetic mouse ovary
P2860
Advanced glycation end-product (AGE) induces apoptosis in human retinal ARPE-19 cells via promoting mitochondrial dysfunction and activating the Fas-FasL signaling.
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年论文
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2015年论文
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2015年论文
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name
Advanced glycation end-product ...... vating the Fas-FasL signaling.
@en
type
label
Advanced glycation end-product ...... vating the Fas-FasL signaling.
@en
prefLabel
Advanced glycation end-product ...... vating the Fas-FasL signaling.
@en
P2093
P2860
P1476
Advanced glycation end-product ...... vating the Fas-FasL signaling.
@en
P2093
Changzheng Chen
Yiqiao Xing
Zhimin Qian
P2860
P304
P356
10.1080/09168451.2015.1095065
P577
2015-10-19T00:00:00Z