JAK-STAT-mediated chronic inflammation impairs cytotoxic T lymphocyte activation to decrease anti-PD-1 immunotherapy efficacy in pancreatic cancer.
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Immune Evasion in Pancreatic Cancer: From Mechanisms to Therapy.Contrasting roles of H3K4me3 and H3K9me3 in regulation of apoptosis and gemcitabine resistance in human pancreatic cancer cells.Targeting the IL-6/JAK/STAT3 signalling axis in cancer.STAT3 as a potential immunotherapy biomarker in oncogene-addicted non-small cell lung cancer.Subgroup analysis reveals molecular heterogeneity and provides potential precise treatment for pancreatic cancers
P2860
JAK-STAT-mediated chronic inflammation impairs cytotoxic T lymphocyte activation to decrease anti-PD-1 immunotherapy efficacy in pancreatic cancer.
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2017 nî lūn-bûn
@nan
2017年の論文
@ja
2017年論文
@yue
2017年論文
@zh-hant
2017年論文
@zh-hk
2017年論文
@zh-mo
2017年論文
@zh-tw
2017年论文
@wuu
2017年论文
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2017年论文
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name
JAK-STAT-mediated chronic infl ...... efficacy in pancreatic cancer.
@en
type
label
JAK-STAT-mediated chronic infl ...... efficacy in pancreatic cancer.
@en
prefLabel
JAK-STAT-mediated chronic infl ...... efficacy in pancreatic cancer.
@en
P2093
P2860
P1433
P1476
JAK-STAT-mediated chronic infl ...... efficacy in pancreatic cancer.
@en
P2093
Asif Talukder
Chunwan Lu
Nagendra Singh
Natasha M Savage
P2860
P304
P356
10.1080/2162402X.2017.1291106
P577
2017-02-10T00:00:00Z