RU486 Induces Pro-Apoptotic Endoplasmic Reticulum Stress Through the Induction of CHOP Expression by Enhancing C/EBPδ Expression in Human Renal Carcinoma Caki Cells.

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TRPV1 channels and the progesterone receptor Sig-1R interact to regulate pain.Z-FL-COCHO, a cathepsin S inhibitor, enhances oxaliplatin-mediated apoptosis through the induction of endoplasmic reticulum stress

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RU486 Induces Pro-Apoptotic Endoplasmic Reticulum Stress Through the Induction of CHOP Expression by Enhancing C/EBPδ Expression in Human Renal Carcinoma Caki Cells.

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RU486 Induces Pro-Apoptotic En ...... an Renal Carcinoma Caki Cells.

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RU486 Induces Pro-Apoptotic En ...... an Renal Carcinoma Caki Cells.

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RU486 Induces Pro-Apoptotic En ...... an Renal Carcinoma Caki Cells.

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RU486 Induces Pro-Apoptotic En ...... an Renal Carcinoma Caki Cells.

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Ji Hoon Jang

http://www.w3.org/2001/XMLSchema#string

Jong-Wook Park

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Kyoung-jin Min

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Shin Kim

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Taeg Kyu Kwon

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P2860

XBP1 mRNA is induced by ATF6 and spliced by IRE1 in response to ER stress to produce a highly active transcription factor

Roles of CHOP/GADD153 in endoplasmic reticulum stress

Endoplasmic reticulum stress: cell life and death decisions.

Stress-induced phosphorylation and activation of the transcription factor CHOP (GADD153) by p38 MAP Kinase

CHOP induces death by promoting protein synthesis and oxidation in the stressed endoplasmic reticulum

Differential regulation of CHOP translation by phosphorylated eIF4E under stress conditions.

Amino acids control mammalian gene transcription: activating transcription factor 2 is essential for the amino acid responsiveness of the CHOP promoter.

Contragestion and other clinical applications of RU 486, an antiprogesterone at the receptor.

Effects of mifepristone on invasive and metastatic potential of human gastric adenocarcinoma cell line MKN-45 in vitro and in vivo.

Induction of the growth arrest and DNA damage-inducible gene GADD153 by cisplatin in vitro and in vivo.

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361-369

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10.1002/JCB.25278

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117

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2015-07-14T00:00:00Z

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26174226

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endoplasmic reticulum

RU486 Induces Pro-Apoptotic Endoplasmic Reticulum Stress Through the Induction of CHOP Expression by Enhancing C/EBPδ Expression in Human Renal Carcinoma Caki Cells.

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RU486 Induces Pro-Apoptotic Endoplasmic Reticulum Stress Through the Induction of CHOP Expression by Enhancing C/EBPδ Expression in Human Renal Carcinoma Caki Cells.

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