A self-enforcing CD44s/ZEB1 feedback loop maintains EMT and stemness properties in cancer cells.
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Pancreatic cancer stem cell markers and exosomes - the incentive pushNew Insights into the Crossroads between EMT and Stemness in the Context of CancerEpithelial-Mesenchymal Transition (EMT) and Regulation of EMT Factors by Steroid Nuclear Receptors in Breast Cancer: A Review and in Silico InvestigationAberrant Splicing of Estrogen Receptor, HER2, and CD44 Genes in Breast CancerTargeting the VEGF-C/VEGFR3 axis suppresses Slug-mediated cancer metastasis and stemness via inhibition of KRAS/YAP1 signalingTackling Cancer Stem Cells via Inhibition of EMT Transcription FactorsThe Role of Fibroblasts in Pancreatic Cancer: Extracellular Matrix Versus Paracrine Factors.Overexpression of NOTCH-regulated Ankyrin Repeat Protein is associated with papillary thyroid carcinoma progression.Expression and Function of CD44 in Epithelial Ovarian CarcinomaElucidation of molecular and functional heterogeneity through differential expression network analyses of discrete tumor subsetsInvestigating the Molecular Basis of PPCD3: Characterization of ZEB1 Regulation of COL4A3 Expression.Mesenchymal-Epithelial Transition in Sarcomas Is Controlled by the Combinatorial Expression of MicroRNA 200s and GRHL2.Virtual screening-driven repositioning of etoposide as CD44 antagonist in breast cancer cellsThe epithelial to mesenchymal transition (EMT) and cancer stem cells: implication for treatment resistance in pancreatic cancerMicroenvironmental networks promote tumor heterogeneity and enrich for metastatic cancer stem-like cells in Luminal-A breast tumor cells.A novel ZEB1/HAS2 positive feedback loop promotes EMT in breast cancerDeciphering Epithelial-Mesenchymal Transition Regulatory Networks in Cancer through Computational ApproachesSerglycin as a potential biomarker for glioma: association of serglycin expression, extent of mast cell recruitment and glioblastoma progression.LKB1 pro-oncogenic activity triggers cell survival in circulating tumor cells.A novel spontaneous model of epithelial-mesenchymal transition (EMT) using a primary prostate cancer derived cell line demonstrating distinct stem-like characteristics.The long non-coding RNA NEAT1 regulates epithelial to mesenchymal transition and radioresistance in through miR-204/ZEB1 axis in nasopharyngeal carcinoma.DNA methylation variations are required for epithelial-to-mesenchymal transition induced by cancer-associated fibroblasts in prostate cancer cells.The GRHL2/ZEB Feedback Loop-A Key Axis in the Regulation of EMT in Breast Cancer.Potent EMT and CSC Phenotypes Are Induced By Oncostatin-M in Pancreatic Cancer.The ESRP1-GPR137 axis contributes to intestinal pathogenesis.MicroRNA-23a promotes pancreatic cancer metastasis by targeting epithelial splicing regulator protein 1.Dual Roles for Epithelial Splicing Regulatory Proteins 1 (ESRP1) and 2 (ESRP2) in Cancer Progression.SNAIL1-mediated downregulation of FOXA proteins facilitates the inactivation of transcriptional enhancer elements at key epithelial genes in colorectal cancer cells.The CD44 standard isoform contributes to radioresistance of pancreatic cancer cells.Molecular targeting of the Aurora-A/SMAD5 oncogenic axis restores chemosensitivity in human breast cancer cells.Epithelial Mesenchymal Transition in Embryonic Development, Tissue Repair and Cancer: A Comprehensive Overview.CD44 Expression Level and Isoform Contributes to Pancreatic Cancer Cell Plasticity, Invasiveness, and Response to Therapy.High expressions of BCL6 and Lewis y antigen are correlated with high tumor burden and poor prognosis in epithelial ovarian cancer.Functional loss of p53 cooperates with the in vivo microenvironment to promote malignant progression of gastric cancers.The prognostic value and clinicopathological significance of CD44 expression in ovarian cancer: a meta-analysis.Mitotic polarization of transcription factors during asymmetric division establishes fate of forming cancer cells.miR-200/375 control epithelial plasticity-associated alternative splicing by repressing the RNA-binding protein Quaking.Group I Paks are essential for epithelial- mesenchymal transition in an Apc-driven model of colorectal cancerStudy on Attenuating Angiogenesis and Epithelial-Mesenchymal Transition (EMT) of Non-Small Cell Lung Carcinoma (NSCLC) by Regulating MAGEC2
P2860
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P2860
A self-enforcing CD44s/ZEB1 feedback loop maintains EMT and stemness properties in cancer cells.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
A self-enforcing CD44s/ZEB1 fe ...... ss properties in cancer cells.
@en
type
label
A self-enforcing CD44s/ZEB1 fe ...... ss properties in cancer cells.
@en
prefLabel
A self-enforcing CD44s/ZEB1 fe ...... ss properties in cancer cells.
@en
P2093
P2860
P50
P356
P1476
A self-enforcing CD44s/ZEB1 fe ...... ess properties in cancer cells
@en
P2093
Bogdan-Tiberius Preca
Jessica Pfannstiel
Karolina Bajdak
Kerstin Mock
Marc P Stemmler
Simone Brabletz
Thomas Brabletz
Ulrich T Hopt
Vignesh Sundararajan
P2860
P304
P356
10.1002/IJC.29642
P577
2015-06-30T00:00:00Z