The bystander cell-killing effect mediated by nitric oxide in normal human fibroblasts varies with irradiation dose but not with radiation quality.

about

Ultra-short laser-accelerated proton pulses have similar DNA-damaging effectiveness but produce less immediate nitroxidative stress than conventional proton beams.The Moss Physcomitrella patens Is Hyperresistant to DNA Double-Strand Breaks Induced by γ-Irradiation.

P2860

Q36118000-4BA8C165-169F-42A3-AD62-5EB1DA315151 Q50133924-AC1D1148-5E2A-43CC-8866-904467BF09F5

P2860

The bystander cell-killing effect mediated by nitric oxide in normal human fibroblasts varies with irradiation dose but not with radiation quality.

Item

http://www.wikidata.org/entity/Q38882929

description

2015 nî lūn-bûn

@nan

2015年の論文

@ja

2015年論文

@yue

2015年論文

@zh-hant

2015年論文

@zh-hk

2015年論文

@zh-mo

2015年論文

@zh-tw

2015年论文

@wuu

2015年论文

@zh

2015年论文

@zh-cn

name

The bystander cell-killing eff ...... ut not with radiation quality.

@en

type

Item

label

The bystander cell-killing eff ...... ut not with radiation quality.

@en

prefLabel

The bystander cell-killing eff ...... ut not with radiation quality.

@en

P1476

The bystander cell-killing eff ...... ut not with radiation quality.

@en

P2093

Hiroko Ikeda

http://www.w3.org/2001/XMLSchema#string

Tomoo Funayama

http://www.w3.org/2001/XMLSchema#string

Yasuhiko Kobayashi

http://www.w3.org/2001/XMLSchema#string

Yasuko Mutou-Yoshihara

http://www.w3.org/2001/XMLSchema#string

P2860

Induction of radioresistance by a nitric oxide-mediated bystander effect.

Biphasic effects of nitric oxide radicals on radiation-induced lethality and chromosome aberrations in human lung cancer cells carrying different p53 gene status.

Gap junction communication and the propagation of bystander effects induced by microbeam irradiation in human fibroblast cultures: the impact of radiation quality.

Radiation-induced bystander effects: past history and future directions.

Role of cell cycle in mediating sensitivity to radiotherapy.

Up-regulation of inducible nitric oxide synthase expression in cancer-prone p53 knockout mice.

Nitric oxide-induced p53 accumulation and regulation of inducible nitric oxide synthase expression by wild-type p53.

Signaling pathways underpinning the manifestations of ionizing radiation-induced bystander effects.

Bystander cell death is modified by sites of energy deposition within cells irradiated with a synchrotron X-ray microbeam.

Induction of bystander response in human glioma cells using high-energy electrons: a role for TGF-beta1.

P304

383-388

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.3109/09553002.2015.1021960

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Yuichiro Yokota

P577

2015-05-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

25908166

http://www.w3.org/2001/XMLSchema#string

The bystander cell-killing effect mediated by nitric oxide in normal human fibroblasts varies with irradiation dose but not with radiation quality.

about

P2860

P2860

The bystander cell-killing effect mediated by nitric oxide in normal human fibroblasts varies with irradiation dose but not with radiation quality.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P698

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P698