Lung inflammation stalls Th17-cell migration en route to the central nervous system during the development of experimental autoimmune encephalomyelitis.

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Tregs restrain dendritic cell autophagy to ameliorate autoimmunity.Helicobacter pylori and gut microbiota in multiple sclerosis versus Alzheimer's disease: 10 pitfalls of microbiome studies.

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Lung inflammation stalls Th17-cell migration en route to the central nervous system during the development of experimental autoimmune encephalomyelitis.

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2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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Lung inflammation stalls Th17- ...... autoimmune encephalomyelitis.

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Lung inflammation stalls Th17- ...... autoimmune encephalomyelitis.

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Lung inflammation stalls Th17- ...... autoimmune encephalomyelitis.

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Lung inflammation stalls Th17- ...... autoimmune encephalomyelitis.

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Keiko Danzaki

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Mari L Shinohara

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Masashi Kanayama

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You-Wen He

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P2860

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NLRP3 inflammasome induces chemotactic immune cell migration to the CNS in experimental autoimmune encephalomyelitis.

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C-C chemokine receptor 6-regulated entry of TH-17 cells into the CNS through the choroid plexus is required for the initiation of EAE.

Autophagy enhances NFκB activity in specific tissue macrophages by sequestering A20 to boost antifungal immunity.

Microtubule-associated protein 1 light chain 3 alpha (LC3)-associated phagocytosis is required for the efficient clearance of dead cells.

Critical role for IL-18 in spontaneous lung inflammation caused by autophagy deficiency.

Interferon-β therapy against EAE is effective only when development of the disease depends on the NLRP3 inflammasome.

Critical regulation of early Th17 cell differentiation by interleukin-1 signaling

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463-469

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scholarly article

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10.1093/INTIMM/DXW013

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5006092

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Lung inflammation stalls Th17-cell migration en route to the central nervous system during the development of experimental autoimmune encephalomyelitis.

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P2860

P2860

Lung inflammation stalls Th17-cell migration en route to the central nervous system during the development of experimental autoimmune encephalomyelitis.

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