β2-AR signaling controls trastuzumab resistance-dependent pathway.
about
Mechanisms Behind the Resistance to Trastuzumab in HER2-Amplified Breast Cancer and Strategies to Overcome ItChronic stress in mice remodels lymph vasculature to promote tumour cell dissemination.Protein kinase A can block EphA2 receptor-mediated cell repulsion by increasing EphA2 S897 phosphorylationTumor suppressor role of miR-3622b-5p in ERBB2-positive cancer.β2-AR activation induces chemoresistance by modulating p53 acetylation through upregulating Sirt1 in cervical cancer cells.Bioorthogonal two-component drug delivery in HER2(+) breast cancer mouse models.Repurposing Drugs in Oncology (ReDO)-Propranolol as an anti-cancer agentMicroRNA Exert Macro Effects on Cancer Bone Metastasis.Serum microRNA-199a/b-3p as a predictive biomarker for treatment response in patients with hepatocellular carcinoma undergoing transarterial chemoembolization.Chronic psychological stress promotes lung metastatic colonization of circulating breast cancer cells by decorating a pre-metastatic niche through activating β-adrenergic signaling.
P2860
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P2860
β2-AR signaling controls trastuzumab resistance-dependent pathway.
description
2015 nî lūn-bûn
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2015年の論文
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年學術文章
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2015年學術文章
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2015年學術文章
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name
β2-AR signaling controls trastuzumab resistance-dependent pathway.
@en
type
label
β2-AR signaling controls trastuzumab resistance-dependent pathway.
@en
prefLabel
β2-AR signaling controls trastuzumab resistance-dependent pathway.
@en
P2093
P2860
P356
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P1476
β2-AR signaling controls trastuzumab resistance-dependent pathway.
@en
P2093
P2860
P2888
P356
10.1038/ONC.2015.58
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P577
2015-03-23T00:00:00Z
P6179
1015222847