GDF15 derived from both tumor-associated macrophages and esophageal squamous cell carcinomas contributes to tumor progression via Akt and Erk pathways.
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An autocrine/paracrine circuit of growth differentiation factor (GDF) 15 has a role for maintenance of breast cancer stem-like cells.Prognostic value of PAX9 in patients with esophageal squamous cell carcinoma and its prediction value to radiation sensitivityGDF15 promotes EMT and metastasis in colorectal cancer.IMP1 suppresses breast tumor growth and metastasis through the regulation of its target mRNAs.Infiltration of tumor-associated macrophages is involved in CD44 expression in clear cell renal cell carcinoma.p-Hydroxylcinnamaldehyde induces the differentiation of oesophageal carcinoma cells via the cAMP-RhoA-MAPK signalling pathway.Collaboration of cancer-associated fibroblasts and tumour-associated macrophages for neuroblastoma development.GDF15 contributes to radioresistance and cancer stemness of head and neck cancer by regulating cellular reactive oxygen species via a SMAD-associated signaling pathway.A GDF15 3' UTR variant, rs1054564, results in allele-specific translational repression of GDF15 by hsa-miR-1233-3pSmall cell lung cancer: Recruitment of macrophages by circulating tumor cells.Software-assisted morphometric and phenotype analyses of human peripheral blood monocyte-derived macrophages induced by a microenvironment model of human esophageal squamous cell carcinoma.CXCL8 derived from tumor-associated macrophages and esophageal squamous cell carcinomas contributes to tumor progression by promoting migration and invasion of cancer cells.Mesenchyme-derived factors enhance preneoplastic growth by non-genotoxic carcinogens in rat liver.New extracellular factors in glioblastoma multiforme development: neurotensin, growth differentiation factor-15, sphingosine-1-phosphate and cytomegalovirus infection.
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P2860
GDF15 derived from both tumor-associated macrophages and esophageal squamous cell carcinomas contributes to tumor progression via Akt and Erk pathways.
description
2015 nî lūn-bûn
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2015年の論文
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
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name
GDF15 derived from both tumor- ...... sion via Akt and Erk pathways.
@en
type
label
GDF15 derived from both tumor- ...... sion via Akt and Erk pathways.
@en
prefLabel
GDF15 derived from both tumor- ...... sion via Akt and Erk pathways.
@en
P2093
P2860
P1476
GDF15 derived from both tumor- ...... ssion via Akt and Erk pathways
@en
P2093
Manabu Shigeoka
Naoki Urakawa
Nobuhisa Takase
Noriaki Arai
Soken Utsunomiya
Yoshihiro Kakeji
Yu-ichiro Koma
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P2888
P304
P356
10.1038/LABINVEST.2015.36
P577
2015-03-02T00:00:00Z
P6179
1022015258