Cell entry of bovine ephemeral fever virus requires activation of Src-JNK-AP1 and PI3K-Akt-NF-κB pathways as well as Cox-2-mediated PGE2 /EP receptor signalling to enhance clathrin-mediated virus endocytosis.
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Important Role of the IL-32 Inflammatory Network in the Host Response against Viral InfectionLPS promotes a monocyte phenotype permissive for human cytomegalovirus immediate-early gene expression upon infection but not reactivation from latencyInfectious Bursal Disease Virus Activates c-Src To Promote α4β1 Integrin-Dependent Viral Entry by Modulating the Downstream Akt-RhoA GTPase-Actin Rearrangement Cascade.The Src kinases Hck, Fgr and Lyn activate Arg to facilitate IgG-mediated phagocytosis and Leishmania infection.
P2860
Cell entry of bovine ephemeral fever virus requires activation of Src-JNK-AP1 and PI3K-Akt-NF-κB pathways as well as Cox-2-mediated PGE2 /EP receptor signalling to enhance clathrin-mediated virus endocytosis.
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Cell entry of bovine ephemeral ...... in-mediated virus endocytosis.
@en
type
label
Cell entry of bovine ephemeral ...... in-mediated virus endocytosis.
@en
prefLabel
Cell entry of bovine ephemeral ...... in-mediated virus endocytosis.
@en
P2093
P2860
P356
P1476
Cell entry of bovine ephemeral ...... in-mediated virus endocytosis.
@en
P2093
Ching-Yuan Cheng
Wei-Ru Huang
P2860
P304
P356
10.1111/CMI.12414
P577
2015-02-04T00:00:00Z