Genome-wide profiling of HPV integration in cervical cancer identifies clustered genomic hot spots and a potential microhomology-mediated integration mechanism.
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Mate-Pair Sequencing as a Powerful Clinical Tool for the Characterization of Cancers with a DNA Viral EtiologyMolecular mechanisms of HPV mediated neoplastic progressionIntronless WNT10B-short variant underlies new recurrent allele-specific rearrangement in acute myeloid leukaemiaClassical risk factors, but not HPV status, predict survival after chemoradiotherapy in advanced head and neck cancer patients.Reduction in the copy number and expression level of the recurrent human papillomavirus integration gene fragile histidine triad (FHIT) predicts the transition of cervical lesions.Possible Human Papillomavirus 38 Contamination of Endometrial Cancer RNA Sequencing Samples in The Cancer Genome Atlas Database.NGS-based approach to determine the presence of HPV and their sites of integration in human cancer genome.Functional variants of human papillomavirus type 16 demonstrate host genome integration and transcriptional alterations corresponding to their unique cancer epidemiology.Universal Human Papillomavirus Typing Assay: Whole-Genome Sequencing following Target EnrichmentFull-length single-cell RNA-seq applied to a viral human cancer: applications to HPV expression and splicing analysis in HeLa S3 cellsGenome Analysis of Latin American Cervical Cancer: Frequent Activation of the PIK3CA PathwayMiR-125a suppresses tumor growth, invasion and metastasis in cervical cancer by targeting STAT3.Detection of human papillomavirus DNA in peri-tumor tissues and pelvic lymph nodes as potential molecular marker of micrometastasis in cervical cancer.DNA Damage Reduces the Quality, but Not the Quantity of Human Papillomavirus 16 E1 and E2 DNA ReplicationGenetic variants within microRNA-binding site of RAD51B are associated with risk of cervical cancer in Chinese women.Dysregulation of host cellular genes targeted by human papillomavirus (HPV) integration contributes to HPV-related cervical carcinogenesis.Genomic and oncogenic preference of HBV integration in hepatocellular carcinoma.Genome-wide profiling of the human papillomavirus DNA integration in cervical intraepithelial neoplasia and normal cervical epithelium by HPV capture technology.HPV16 integration probably contributes to cervical oncogenesis through interrupting tumor suppressor genes and inducing chromosome instability.Analysis of E2 gene integrity in HPV16 and HPV58 viruses isolated from women with cervical pathology.Proteomics-based identification of VDAC1 as a tumor promoter in cervical carcinomaPancancer modelling predicts the context-specific impact of somatic mutations on transcriptional programs.Next generation sequencing and its applications in HPV-associated cancers.Decreased expression of semaphorin 3D is associated with genesis and development in colorectal cancer.HPV positive neuroendocrine cervical cancer cells are dependent on Myc but not E6/E7 viral oncogenes.High Risk Alpha Papillomavirus Oncogenes Impair the Homologous Recombination Pathway.Molecular mechanisms of human papillomavirus-related carcinogenesis in head and neck cancer.Identification of human papillomavirus (HPV) 16 DNA integration and the ensuing patterns of methylation in HPV-associated head and neck squamous cell carcinoma cell lines.Aberrant PD-L1 expression through 3'-UTR disruption in multiple cancers.Mechanistic and Technical Challenges in Studying the Human Microbiome and Cancer EpidemiologyThe molecular mechanism of human papillomavirus-induced carcinogenesis in head and neck squamous cell carcinoma.Genomic characterization of viral integration sites in HPV-related cancers.The variable clinicopathological categories and role of human papillomavirus in cervical adenocarcinoma: A hospital based nation-wide multi-center retrospective study across China.HPV Integration in Head and Neck Squamous Cell Carcinomas: Cause and Consequence.Molecular approaches for HPV genotyping and HPV-DNA physical status.Current and Emerging Molecular Tests for Human Papillomavirus-Related Neoplasia in the Genomic Era.A novel mechanism of rs763110 polymorphism contributing to cervical cancer risk by affecting the binding affinity of C/EBPβ and OCT1 complex to chromatin.Fragile sites in cancer: more than meets the eye.DNA Oncogenic Virus-Induced Oxidative Stress, Genomic Damage, and Aberrant Epigenetic AlterationsSomatic Host Cell Alterations in HPV Carcinogenesis.
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Genome-wide profiling of HPV integration in cervical cancer identifies clustered genomic hot spots and a potential microhomology-mediated integration mechanism.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年学术文章
@wuu
2015年学术文章
@zh-cn
2015年学术文章
@zh-hans
2015年学术文章
@zh-my
2015年学术文章
@zh-sg
2015年學術文章
@yue
2015年學術文章
@zh
2015年學術文章
@zh-hant
name
Genome-wide profiling of HPV i ...... ediated integration mechanism.
@en
type
label
Genome-wide profiling of HPV i ...... ediated integration mechanism.
@en
prefLabel
Genome-wide profiling of HPV i ...... ediated integration mechanism.
@en
P2093
P2860
P50
P356
P1433
P1476
Genome-wide profiling of HPV i ...... ediated integration mechanism.
@en
P2093
Changlin Zhang
Changyu Wang
Hongjie Liu
Jianfeng Zhou
P2860
P2888
P304
P356
10.1038/NG.3178
P407
P577
2015-01-12T00:00:00Z