FBXW7-Induced MTOR Degradation Forces Autophagy to Counteract Persistent Prion Infection.
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Overexpression of PLK3 Mediates the Degradation of Abnormal Prion Proteins Dependent on Chaperone-Mediated AutophagyStrain-Dependent Effect of Macroautophagy on Abnormally Folded Prion Protein Degradation in Infected Neuronal Cells.Activation of the AMPK-ULK1 pathway plays an important role in autophagy during prion infection.
P2860
FBXW7-Induced MTOR Degradation Forces Autophagy to Counteract Persistent Prion Infection.
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2015年の論文
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name
FBXW7-Induced MTOR Degradation Forces Autophagy to Counteract Persistent Prion Infection.
@en
type
label
FBXW7-Induced MTOR Degradation Forces Autophagy to Counteract Persistent Prion Infection.
@en
prefLabel
FBXW7-Induced MTOR Degradation Forces Autophagy to Counteract Persistent Prion Infection.
@en
P2093
P2860
P1476
FBXW7-Induced MTOR Degradation Forces Autophagy to Counteract Persistent Prion Infection.
@en
P2093
Xiao-Ping Dong
Xue-Yu Fan
P2860
P2888
P304
P356
10.1007/S12035-014-9028-7
P577
2015-01-13T00:00:00Z
P6179
1026642461