Propofol Administration During Early Postnatal Life Suppresses Hippocampal Neurogenesis. - Wikidata - wikimedia - TriplyDB

Propofol Administration During Early Postnatal Life Suppresses Hippocampal Neurogenesis.

Propofol Administration During Early Postnatal Life Suppresses Hippocampal Neurogenesis.

http://www.wikidata.org/entity/Q38921291

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Propofol-induced downregulation of NR2B membrane translocation in hippocampus and spatial memory deficits of neonatal mice.Effect of propofol on microRNA expression in rat primary embryonic neural stem cells Grafted c-kit+/SSEA1- eye-wall progenitor cells delay retinal degeneration in mice by regulating neural plasticity and forming new graft-to-host synapses.A holistic approach to anesthesia-induced neurotoxicity and its implications for future mechanistic studies.Recent Insights Into Molecular Mechanisms of Propofol-Induced Developmental Neurotoxicity: Implications for the Protective Strategies.Neurogenesis and developmental anesthetic neurotoxicity.Liver X Receptor β Is Involved in Formalin-Induced Spontaneous Pain.Propofol's Effects on the Fetal Brain for Non-Obstetric Surgery.Propofol Exposure in Early Life Induced Developmental Impairments in the Mouse Cerebellum.Propofol Affects Neurodegeneration and Neurogenesis by Regulation of Autophagy via Effects on Intracellular Calcium Homeostasis.Upregulation of Cdh1 Attenuates Isoflurane-Induced Neuronal Apoptosis and Long-Term Cognitive Impairments in Developing Rats.Hypermethylation of Hippocampal Synaptic Plasticity-Related genes is Involved in Neonatal Sevoflurane Exposure-Induced Cognitive Impairments in Rats.Silica nanoparticle exposure during the neonatal period impairs hippocampal precursor proliferation and social behavior later in life.Dexmedetomidine attenuates the propofol-induced long-term neurotoxicity in the developing brain of rats by enhancing the PI3K/Akt signaling pathway Propofol Alters Long Non-Coding RNA Profiles in the Neonatal Mouse Hippocampus: Implication of Novel Mechanisms in Anesthetic-Induced Developmental Neurotoxicity

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Propofol Administration During Early Postnatal Life Suppresses Hippocampal Neurogenesis.

http://www.wikidata.org/entity/Q38921291

description

2015 nî lūn-bûn

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2015年の論文

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年學術文章

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2015年學術文章

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2015年學術文章

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Propofol Administration During Early Postnatal Life Suppresses Hippocampal Neurogenesis.

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Propofol Administration During Early Postnatal Life Suppresses Hippocampal Neurogenesis.

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Propofol Administration During Early Postnatal Life Suppresses Hippocampal Neurogenesis.

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Molecular Neurobiology

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Propofol Administration During Early Postnatal Life Suppresses Hippocampal Neurogenesis.

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Hong Li

http://www.w3.org/2001/XMLSchema#string

Jing Huang

http://www.w3.org/2001/XMLSchema#string

Sheng Jing

http://www.w3.org/2001/XMLSchema#string

Tiande Yang

http://www.w3.org/2001/XMLSchema#string

Xi Chen

http://www.w3.org/2001/XMLSchema#string

Xiaohang Bao

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Xiaotang Fan

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Zhiyong Du

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P2860

Multipotent neural cell lines can engraft and participate in development of mouse cerebellum

Propofol directly increases tau phosphorylation

Antisense Noggin oligodeoxynucleotide administration decreases cell proliferation in the dentate gyrus of adult rats

Adult neurogenesis in the mammalian central nervous system

New neurons and new memories: how does adult hippocampal neurogenesis affect learning and memory?

Propofol-induced changes in neurotrophic signaling in the developing nervous system in vivo

Moderate traumatic brain injury causes acute dendritic and synaptic degeneration in the hippocampal dentate gyrus.

Hippocampal neurogenesis and dendritic plasticity support running-improved spatial learning and depression-like behaviour in stressed rats

General anesthetics in pediatric anesthesia: influences on the developing brain.

Molecular and cellular mechanisms of general anaesthesia.

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