AID expression in B-cell lymphomas causes accumulation of genomic uracil and a distinct AID mutational signature.
about
Integrative systems medicine approaches to identify molecular targets in lymphoid malignanciesIsotype-switched follicular lymphoma displays dissociation between activation-induced cytidine deaminase expression and somatic hypermutationBase changes in tumour DNA have the power to reveal the causes and evolution of cancer.Mutagenesis by AID: Being in the Right Place at the Right Time.Whole-genome sequencing reveals activation-induced cytidine deaminase signatures during indolent chronic lymphocytic leukaemia evolutionAID-associated DNA repair pathways regulate malignant transformation in a murine model of BCL6-driven diffuse large B-cell lymphoma.Clusters of Multiple Mutations: Incidence and Molecular MechanismsDetection of uracil within DNA using a sensitive labeling method for in vitro and cellular applications.PARP activation promotes nuclear AID accumulation in lymphoma cellsExcerpts from the 1st international NTNU symposium on current and future clinical biomarkers of cancer: innovation and implementation, June 16th and 17th 2016, Trondheim, Norway.Proteome alterations associated with transformation of multiple myeloma to secondary plasma cell leukemia.Mechanisms and clinical applications of chromosomal instability in lymphoid malignancy.Parent-of-origin-specific signatures of de novo mutations.The Complex Interplay between DNA Injury and Repair in Enzymatically Induced Mutagenesis and DNA Damage in B Lymphocytes.A novel class of chemicals that react with abasic sites in DNA and specifically kill B cell cancers.Higher Expression of Activation-induced Cytidine Deaminase Is Significantly Associated with Merkel Cell Polyomavirus-negative Merkel Cell CarcinomasNon-coding cancer driver candidates identified with a sample- and position-specific model of the somatic mutation rate.*K-means and cluster models for cancer signatures.High-risk follicular lymphomas harbour more somatic mutations including those in the AID-motifLiving Organisms Author Their Read-Write Genomes in Evolution.Biochemical Regulatory Features of Activation-Induced Cytidine Deaminase Remain Conserved from Lampreys to Humans.Diagnostic value of STMN1, LMO2, HGAL, AID expression and 1p36 chromosomal abnormalities in primary cutaneous B cell lymphomas.Investigation of AID, Dicer, and Drosha Expressions in Patients with Chronic Lymphocytic Leukemia.Novel UCHL1 mutations reveal new insights into ubiquitin processing.Genetic heterogeneity and mutational signature in Chinese Epstein-Barr virus-positive diffuse large B-cell lymphoma
P2860
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P2860
AID expression in B-cell lymphomas causes accumulation of genomic uracil and a distinct AID mutational signature.
description
2014 nî lūn-bûn
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2014年の論文
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2014年学术文章
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2014年学术文章
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2014年学术文章
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2014年学术文章
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2014年学术文章
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2014年學術文章
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2014年學術文章
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name
AID expression in B-cell lymph ...... inct AID mutational signature.
@en
type
label
AID expression in B-cell lymph ...... inct AID mutational signature.
@en
prefLabel
AID expression in B-cell lymph ...... inct AID mutational signature.
@en
P2093
P50
P1433
P1476
AID expression in B-cell lymph ...... tinct AID mutational signature
@en
P2093
Anastasia Galashevskaya
Berit Doseth
Bodil Kavli
Henrik Sahlin Pettersen
Mirta M L Sousa
Nina-Beate Liabakk
Per Arne Aas
P356
10.1016/J.DNAREP.2014.11.006
P577
2014-11-24T00:00:00Z