Molecular analysis of sunitinib resistant renal cell carcinoma cells after sequential treatment with RAD001 (everolimus) or sorafenib.
about
Infiltrating mast cells promote renal cell carcinoma angiogenesis by modulating PI3K→︀AKT→︀GSK3β→︀AM signaling.RIP1-dependent reactive oxygen species production executes artesunate-induced cell death in renal carcinoma Caki cells.Adrenomedullin blockade suppresses sunitinib-resistant renal cell carcinoma growth by targeting the ERK/MAPK pathway.Intra-tumour molecular heterogeneity of clear cell renal cell carcinoma reveals the diversity of the response to targeted therapies using patient-derived xenograft models.Everolimus (RAD001) inhibits the proliferation of rat vascular smooth muscle cells by up-regulating the activity of the p27/kip1 gene promoter.LncRNA RP11-436H11.5, functioning as a competitive endogenous RNA, upregulates BCL-W expression by sponging miR-335-5p and promotes proliferation and invasion in renal cell carcinoma.Use of mammalian target of rapamycin inhibitors after failure of tyrosine kinase inhibitors in patients with metastatic renal cell carcinoma undergoing hemodialysis: A single-center experience with four cases.
P2860
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P2860
Molecular analysis of sunitinib resistant renal cell carcinoma cells after sequential treatment with RAD001 (everolimus) or sorafenib.
description
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name
Molecular analysis of sunitini ...... 001 (everolimus) or sorafenib.
@en
type
label
Molecular analysis of sunitini ...... 001 (everolimus) or sorafenib.
@en
prefLabel
Molecular analysis of sunitini ...... 001 (everolimus) or sorafenib.
@en
P2093
P2860
P356
P1476
Molecular analysis of sunitini ...... 001 (everolimus) or sorafenib.
@en
P2093
Axel Haferkamp
Eva Juengel
Georg Bartsch
Igor Tsaur
Jasmina Makarević
Michael Reiter
Roman A Blaheta
P2860
P304
P356
10.1111/JCMM.12471
P577
2014-12-02T00:00:00Z