Gene-environment interactions in development and disease.

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Ethanol itself is a holoprosencephaly-inducing teratogen.Embryonic Ethanol Exposure Affects Early- and Late-Added Cardiac Precursors and Produces Long-Lasting Heart Chamber Defects in Zebrafish.

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Gene-environment interactions in development and disease.

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2016 nî lūn-bûn

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2016年の論文

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2016年論文

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2016年論文

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2016年论文

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2016年论文

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name

Gene-environment interactions in development and disease.

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Gene-environment interactions in development and disease.

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Gene-environment interactions in development and disease.

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Gene-environment interactions in development and disease.

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P2093

C Lovely

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Johann Eberhart

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Mindy Rampersad

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Yohaan Fernandes

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P2860

Substantial contribution of extrinsic risk factors to cancer development

Neurotoxic effects and biomarkers of lead exposure: a review

Evidence for gene-environment interaction in a genome wide study of nonsyndromic cleft palate

The heterozygote advantage in phenylketonuria

ApoE genotype, past adult lead exposure, and neurobehavioral function.

Lipids and Oxidative Stress Associated with Ethanol-Induced Neurological Damage

Advances in Diagnosis and Treatment of Fetal Alcohol Spectrum Disorders: From Animal Models to Human Studies

Dysregulation of microRNA expression and function contributes to the etiology of fetal alcohol spectrum disorders

Regulation of mTORC1 by PI3K signaling

Trace elements as paradigms of developmental neurotoxicants: Lead, methylmercury and arsenic

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scholarly article

P356

10.1002/WDEV.247

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2016-09-14T00:00:00Z

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27626243

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5191946

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Gene-environment interactions in development and disease.

about

P2860

P2860

Gene-environment interactions in development and disease.

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