Leishmania donovani infection initiates T cell-independent chemokine responses, which are subsequently amplified in a T cell-dependent manner.
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In vivo involvement of polymorphonuclear neutrophils in Leishmania infantum infectionDynamic imaging of experimental Leishmania donovani-induced hepatic granulomas detects Kupffer cell-restricted antigen presentation to antigen-specific CD8 T cellsLessons from other diseases: granulomatous inflammation in leishmaniasisRegulation of immunity during visceral Leishmania infectionMononuclear cell recruitment, granuloma assembly, and response to treatment in experimental visceral leishmaniasis: intracellular adhesion molecule 1-dependent and -independent regulationClinical and experimental advances in treatment of visceral leishmaniasis.Enhanced hematopoietic activity accompanies parasite expansion in the spleen and bone marrow of mice infected with Leishmania donovaniLeishmania donovani-induced expression of signal regulatory protein alpha on Kupffer cells enhances hepatic invariant NKT-cell activationInvariant NKT cells drive hepatic cytokinic microenvironment favoring efficient granuloma formation and early control of Leishmania donovani infection.Higher expression of CCL2, CCL4, CCL5, CCL21, and CXCL8 chemokines in the skin associated with parasite density in canine visceral leishmaniasis.Tissue granuloma structure-function in experimental visceral leishmaniasis.Balancing immunity and pathology in visceral leishmaniasis.Immunoenhancement combined with amphotericin B as treatment for experimental visceral leishmaniasis.CXCL10/gamma interferon-inducible protein 10-mediated protection against Leishmania amazonensis infection in miceChemokine expression dynamics in mycobacterial (type-1) and schistosomal (type-2) antigen-elicited pulmonary granuloma formation.Effects of CXCL10 on dendritic cell and CD4+ T-cell functions during Leishmania amazonensis infection.Chemokine gene expression in toll-like receptor-competent and -deficient mice infected with Leishmania major.Role of chemokines in regulation of immunity against leishmaniasis.Immunotherapy as a strategy for treatment of leishmaniasis: a review of the literature.Involvement and interactions of different immune cells and their cytokines in human visceral leishmaniasis.Nanoliposomal Buparvaquone Immunomodulates Leishmania infantum-Infected Macrophages and Is Highly Effective in a Murine Model.Invariant NKT cells are essential for the regulation of hepatic CXCL10 gene expression during Leishmania donovani infection.Transgenic T cell-specific expression of CXCR3 enhances splenic and hepatic T cell accumulation but does not affect the outcome of visceral leishmaniasis.Monocyte chemotactic protein-1 stimulates the killing of leishmania major by human monocytes, acts synergistically with IFN-gamma and is antagonized by IL-4.F₁ cross-breed between susceptible BALB/c and resistant Swiss mice infected with Leishmania major exhibit an intermediate phenotype for lesion sizes and type 1 cytokines but show low level of total IgG antibodies.Macrophage Transactivation for Chemokine Production Identified as a Negative Regulator of Granulomatous Inflammation Using Agent-Based Modeling.
P2860
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P2860
Leishmania donovani infection initiates T cell-independent chemokine responses, which are subsequently amplified in a T cell-dependent manner.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
1999年论文
@zh
1999年论文
@zh-cn
name
Leishmania donovani infection ...... in a T cell-dependent manner.
@en
type
label
Leishmania donovani infection ...... in a T cell-dependent manner.
@en
prefLabel
Leishmania donovani infection ...... in a T cell-dependent manner.
@en
P2093
P2860
P1476
Leishmania donovani infection ...... in a T cell-dependent manner.
@en
P2093
Cotterell SE
Engwerda CR
P2860
P304
P356
10.1002/(SICI)1521-4141(199901)29:01<203::AID-IMMU203>3.0.CO;2-B
P407
P577
1999-01-01T00:00:00Z