GRP78 promotes the invasion of pancreatic cancer cells by FAK and JNK. - Wikidata - wikimedia - TriplyDB

GRP78 promotes the invasion of pancreatic cancer cells by FAK and JNK.

GRP78 promotes the invasion of pancreatic cancer cells by FAK and JNK.

http://www.wikidata.org/entity/Q38956837

about

FOXM1 promotes invasion and migration of colorectal cancer cells partially dependent on HSPA5 transactivation Mining for Candidate Genes Related to Pancreatic Cancer Using Protein-Protein Interactions and a Shortest Path Approach.GRP78 confers the resistance to 5-FU by activating the c-Src/LSF/TS axis in hepatocellular carcinoma Neuregulin1-β decreases interleukin-1β-induced RhoA activation, myosin light chain phosphorylation, and endothelial hyperpermeability.High expression of glucose-regulated protein 78 (GRP78) is associated with metastasis and poor prognosis in patients with esophageal squamous cell carcinoma.PAK4 suppresses PDZ-RhoGEF activity to drive invadopodia maturation in melanoma cells.c-Jun N-terminal kinase in pancreatic tumor stroma augments tumor development in mice.Secreted GRP78 activates EGFR-SRC-STAT3 signaling and confers the resistance to sorafeinib in HCC cells.Role of matrix metalloproteinase-2/9 (MMP2/9) in lead-induced changes in an in vitro blood-brain barrier model.Re-expression of Sall1 in podocytes protects against adriamycin-induced nephrosis.Identification of PGAM1 as a putative therapeutic target for pancreatic ductal adenocarcinoma metastasis using quantitative proteomics.New Hope for Pancreatic Ductal Adenocarcinoma Treatment Targeting Endoplasmic Reticulum Stress Response: A Systematic Review

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P2860

GRP78 promotes the invasion of pancreatic cancer cells by FAK and JNK.

http://www.wikidata.org/entity/Q38956837

description

2014 nî lūn-bûn

@nan

2014年の論文

@ja

2014年論文

@yue

2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

2014年论文

@zh

2014年论文

@zh-cn

name

GRP78 promotes the invasion of pancreatic cancer cells by FAK and JNK.

@en

type

label

GRP78 promotes the invasion of pancreatic cancer cells by FAK and JNK.

@en

prefLabel

GRP78 promotes the invasion of pancreatic cancer cells by FAK and JNK.

@en

P1433

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Molecular and Cellular Biochemistry

P1476

GRP78 promotes the invasion of pancreatic cancer cells by FAK and JNK.

@en

P2093

J P Zhou

http://www.w3.org/2001/XMLSchema#string

Ming Dong

http://www.w3.org/2001/XMLSchema#string

X P Yuan

http://www.w3.org/2001/XMLSchema#string

Xin Li

http://www.w3.org/2001/XMLSchema#string

P2860

Invasion and metastasis in pancreatic cancer

The unfolded protein response regulator GRP78 is a novel predictive biomarker in colorectal cancer.

IGFBP-3 binds GRP78, stimulates autophagy and promotes the survival of breast cancer cells exposed to adverse microenvironments

Integrins in cell migration--the actin connection.

Grp78 promotes the invasion of hepatocellular carcinoma.

Protein Signatures in Human MDA-MB-231 Breast Cancer Cells Indicating a More Invasive Phenotype Following Knockdown of Human Endometase/Matrilysin-2 by siRNA.

Protein disulfide isomerase-mediated disulfide bonds regulate the gelatinolytic activity and secretion of matrix metalloproteinase-9.

The glucose-regulated proteins: stress induction and clinical applications.

A peptide derived from phage display library exhibits anti-tumor activity by targeting GRP78 in gastric cancer multidrug resistance cells.

Glucose-regulated protein 78 controls cross-talk between apoptosis and autophagy to determine antiestrogen responsiveness

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s11010-014-2204-2

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55-62

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scholarly article

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10.1007/S11010-014-2204-2

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1-2

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398

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2014-09-14T00:00:00Z

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25218495

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