Leigh syndrome in Drosophila melanogaster: morphological and biochemical characterization of Surf1 post-transcriptional silencing
about
F-ATPase of Drosophila melanogaster forms 53-picosiemen (53-pS) channels responsible for mitochondrial Ca2+-induced Ca2+ release.Ligand-Bound GeneSwitch Causes Developmental Aberrations in Drosophila that Are Alleviated by the Alternative OxidaseNutritional Interventions for Mitochondrial OXPHOS Deficiencies: Mechanisms and Model Systems.Feeding difficulties, a key feature of the Drosophila NDUFS4 mitochondrial disease model.SURF1 knockout cloned pigs: Early onset of a severe lethal phenotype.
P2860
Leigh syndrome in Drosophila melanogaster: morphological and biochemical characterization of Surf1 post-transcriptional silencing
description
2014 nî lūn-bûn
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2014年の論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年论文
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2014年论文
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2014年论文
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name
Leigh syndrome in Drosophila m ...... post-transcriptional silencing
@en
type
label
Leigh syndrome in Drosophila m ...... post-transcriptional silencing
@en
prefLabel
Leigh syndrome in Drosophila m ...... post-transcriptional silencing
@en
P2093
P2860
P50
P356
P1476
Leigh syndrome in Drosophila m ...... post-transcriptional silencing
@en
P2093
Caterina Da-Rè
Caterina Millino
Cristiano De Pittà
Mauro A Zordan
Paola Cisotto
Rodolfo Costa
Sophia von Stockum
P2860
P304
29235-29246
P356
10.1074/JBC.M114.602938
P407
P577
2014-08-27T00:00:00Z