Pre-clinical analysis of changes in intra-cellular biochemistry of glioblastoma multiforme (GBM) cells due to c-Myc silencing.
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Inhibition of c-MYC with involvement of ERK/JNK/MAPK and AKT pathways as a novel mechanism for shikonin and its derivatives in killing leukemia cells.The BET inhibitor JQ1 selectively impairs tumour response to hypoxia and downregulates CA9 and angiogenesis in triple negative breast cancer.Epigenetics in cancer stem cells.Alisertib induces G2/M arrest, apoptosis, and autophagy via PI3K/Akt/mTOR- and p38 MAPK-mediated pathways in human glioblastoma cellsUbenimex enhances Brd4 inhibition by suppressing HEXIM1 autophagic degradation and suppressing the Akt pathway in glioma cells.Bisphosphonate-Functionalized Hydroxyapatite Nanoparticles for the Delivery of the Bromodomain Inhibitor JQ1 in the Treatment of Osteosarcoma.Nardostachys jatamansi Targets BDNF-TrkB to Alleviate Ketamine-Induced Schizophrenia-Like Symptoms in Rats.
P2860
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P2860
Pre-clinical analysis of changes in intra-cellular biochemistry of glioblastoma multiforme (GBM) cells due to c-Myc silencing.
description
2014 nî lūn-bûn
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name
Pre-clinical analysis of chang ...... cells due to c-Myc silencing.
@en
type
label
Pre-clinical analysis of chang ...... cells due to c-Myc silencing.
@en
prefLabel
Pre-clinical analysis of chang ...... cells due to c-Myc silencing.
@en
P2093
P2860
P1476
Pre-clinical analysis of chang ...... cells due to c-Myc silencing.
@en
P2093
James E Bradner
Muthukumar Vaidyanathan
Vanisree Arambakkam Janardhanam
Vishal Rajagopalan
P2860
P2888
P304
P356
10.1007/S10571-014-0083-4
P577
2014-07-24T00:00:00Z
P5875
P6179
1026815916