S100A8, S100A9 and S100A12 activate airway epithelial cells to produce MUC5AC via extracellular signal-regulated kinase and nuclear factor-κB pathways.
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NF-kappaB Signaling in Chronic Inflammatory Airway DiseaseRespiratory epithelial cells orchestrate pulmonary innate immunity.Blocking the Interactions between Calcium-Bound S100A12 Protein and the V Domain of RAGE Using TranilastRelevance Rank Platform (RRP) for Functional Filtering of High Content Protein-Protein Interaction Data.Role of inflammasome activation in development and exacerbation of asthma.The effects of in utero vitamin D deficiency on airway smooth muscle mass and lung function.The Human Antimicrobial Protein Calgranulin C Participates in Control of Helicobacter pylori Growth and Regulation of Virulence.Decreased S100A9 Expression Promoted Rat Airway Smooth Muscle Cell Proliferation by Stimulating ROS Generation and Inhibiting p38 MAPK.Do Aging Factors Influence the Clinical Presentation and Management of Chronic Rhinosinusitis?Chronic inflammatory airway diseases: the central role of the epithelium revisited.Transcription Factor NF-κB: An Update on Intervention Strategies.MUC5AC interactions with integrin β4 enhances the migration of lung cancer cells through FAK signaling.All the "RAGE" in lung disease: The receptor for advanced glycation endproducts (RAGE) is a major mediator of pulmonary inflammatory responses.Impaired response of the bronchial epithelium to inflammation characterizes severe equine asthma.Association of S100 calcium-binding protein A12, receptor for advanced glycation endproducts, and nuclear factor-κB expression with inflammation in pulp tissues from tooth caries.IL-4-induced caveolin-1-containing lipid rafts aggregation contributes to MUC5AC synthesis in bronchial epithelial cells.S100A8+ stroma cells predict a good prognosis and inhibit aggressiveness in colorectal carcinoma.Zinc Deficiency via a Splice Switch in Zinc Importer ZIP2/SLC39A2 Causes Cystic Fibrosis-Associated MUC5AC Hypersecretion in Airway Epithelial Cells.Effects of 1,25-Dihydroxyvitamin D3 on the Prevention of Chronic Obstructive Pulmonary Disease (COPD) in Rats Exposed to Air Pollutant Particles Less than 2.5 Micrometers in Diameter (PM2.5).Blocking the ERK1/2 signal pathway can inhibit S100A12 induced human aortic smooth muscle cells damage.S100 Proteins As an Important Regulator of Macrophage Inflammation.Coevolution of the Toll-Like Receptor 4 Complex with Calgranulins and Lipopolysaccharide.Neutrophil extracellular traps promote lipopolysaccharide-induced airway inflammation and mucus hypersecretion in mice.S100A12 as a marker of worse cardiac output and mortality in pulmonary hypertension.Anti-Allergic Inflammatory Activity of Interleukin-37 Is Mediated by Novel Signaling Cascades in Human Eosinophils.Toll-Like Receptor Agonists Modulate Wound Regeneration in Airway Epithelial Cells
P2860
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P2860
S100A8, S100A9 and S100A12 activate airway epithelial cells to produce MUC5AC via extracellular signal-regulated kinase and nuclear factor-κB pathways.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年学术文章
@wuu
2015年学术文章
@zh-cn
2015年学术文章
@zh-hans
2015年学术文章
@zh-my
2015年学术文章
@zh-sg
2015年學術文章
@yue
2015年學術文章
@zh
2015年學術文章
@zh-hant
name
S100A8, S100A9 and S100A12 act ...... nd nuclear factor-κB pathways.
@en
type
label
S100A8, S100A9 and S100A12 act ...... nd nuclear factor-κB pathways.
@en
prefLabel
S100A8, S100A9 and S100A12 act ...... nd nuclear factor-κB pathways.
@en
P2093
P2860
P356
P1433
P1476
S100A8, S100A9 and S100A12 act ...... nd nuclear factor-κB pathways.
@en
P2093
Il Yup Chung
Jin Hyun Kang
Sae Mi Hwang
P2860
P356
10.1111/IMM.12352
P577
2015-01-01T00:00:00Z