Whole transcriptome sequencing identifies increased CXCR2 expression in PNH granulocytes.

about

Whole transcriptome sequencing identifies increased CXCR2 expression in PNH granulocytes.

Item

http://www.wikidata.org/entity/Q38984534

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年学术文章

@wuu

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

@yue

2017年學術文章

@zh

2017年學術文章

@zh-hant

name

Whole transcriptome sequencing identifies increased CXCR2 expression in PNH granulocytes.

@en

type

Item

label

Whole transcriptome sequencing identifies increased CXCR2 expression in PNH granulocytes.

@en

prefLabel

Whole transcriptome sequencing identifies increased CXCR2 expression in PNH granulocytes.

@en

P1476

Whole transcriptome sequencing identifies increased CXCR2 expression in PNH granulocytes

@en

P2093

Angelique Biancotto

http://www.w3.org/2001/XMLSchema#string

Danielle M Townsley

http://www.w3.org/2001/XMLSchema#string

Keyvan Keyvanfar

http://www.w3.org/2001/XMLSchema#string

Neal S Young

http://www.w3.org/2001/XMLSchema#string

Sachiko Kajigaya

http://www.w3.org/2001/XMLSchema#string

Wangmin Qiao

http://www.w3.org/2001/XMLSchema#string

Xingmin Feng

http://www.w3.org/2001/XMLSchema#string

Yanling Xie

http://www.w3.org/2001/XMLSchema#string

P2860

Current concepts in the pathophysiology and treatment of aplastic anemia

Deficiency of the GPI anchor caused by a somatic mutation of the PIG-A gene in paroxysmal nocturnal hemoglobinuria

MIF is a noncognate ligand of CXC chemokine receptors in inflammatory and atherogenic cell recruitment

CXCR2 and CXCR4 antagonistically regulate neutrophil trafficking from murine bone marrow

Deep sequencing reveals stepwise mutation acquisition in paroxysmal nocturnal hemoglobinuria.

The interleukin-8 pathway in cancer.

IL8-CXCR2 pathway inhibition as a therapeutic strategy against MDS and AML stem cells

CXCR2 and CXCL4 regulate survival and self-renewal of hematopoietic stem/progenitor cells.

Differential gene expression in hematopoietic progenitors from paroxysmal nocturnal hemoglobinuria patients reveals an apoptosis/immune response in 'normal' phenotype cells.

Circulating primitive stem cells in paroxysmal nocturnal hemoglobinuria (PNH) are predominantly normal in phenotype but granulocyte colony-stimulating factor treatment mobilizes mainly PNH stem cells.

P304

136-141

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1111/BJH.14502

http://www.w3.org/2001/XMLSchema#string

P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

177

http://www.w3.org/2001/XMLSchema#string

P50

Kohei Hosokawa

P577

2017-02-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

28151558

http://www.w3.org/2001/XMLSchema#string

P921

transcriptome

P932

5378616

http://www.w3.org/2001/XMLSchema#string