Extracellular matrix stiffness and composition jointly regulate the induction of malignant phenotypes in mammary epithelium. - Wikidata - wikimedia - TriplyDB

Extracellular matrix stiffness and composition jointly regulate the induction of malignant phenotypes in mammary epithelium.

Extracellular matrix stiffness and composition jointly regulate the induction of malignant phenotypes in mammary epithelium.

http://www.wikidata.org/entity/Q38984674

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Neural regulation of cancer: from mechanobiology to inflammation The missing link: does tunnelling nanotube-based supercellularity provide a new understanding of chronic and lifestyle diseases?A recapitulative three-dimensional model of breast carcinoma requires perfusion for multi-week growth Cell Invasion Dynamics into a Three Dimensional Extracellular Matrix Fibre Network Composite alginate gels for tunable cellular microenvironment mechanics.Tension in Cancer Processing Techniques and Applications of Silk Hydrogels in Bioengineering Cellular mechano-environment regulates the mammary circadian clock Impact of the physical microenvironment on tumor progression and metastasis Integrins as architects of cell behavior Tissue stiffness dictates development, homeostasis, and disease progression.Drug-triggered and cross-linked self-assembling nanofibrous hydrogels.Thermoreversible and Injectable ABC Polypeptoid Hydrogels: Controlling the Hydrogel Properties through Molecular Design.Quiescence of human muscle stem cells is favored by culture on natural biopolymeric films.Microscale Bioadhesive Hydrogel Arrays for Cell Engineering Applications Ultra-responsive soft matter from strain-stiffening hydrogels.Three-dimensional, soft neotissue arrays as high throughput platforms for the interrogation of engineered tissue environments.Decreasing matrix modulus of PEG hydrogels induces a vascular phenotype in human cord blood stem cells Identifying Triple-Negative Breast Cancer Using Background Parenchymal Enhancement Heterogeneity on Dynamic Contrast-Enhanced MRI: A Pilot Radiomics Study Mechanically patterning the embryonic airway epithelium Tumor-induced remote ECM network orientation steers angiogenesis.Evaluation of cell and matrix mechanics using fluorescence excitation spectroscopy: Feasibility study in collagen gels containing fibroblasts.SAMA: A Method for 3D Morphological Analysis.Spatial Control of Cell-Nanosurface Interactions by Tantalum Oxide Nanodots for Improved Implant Geometry Quantification of regenerative potential in primary human mammary epithelial cells.Matrix rigidity regulates the transition of tumor cells to a bone-destructive phenotype through integrin β3 and TGF-β receptor type II.Hydrogel Pore-Size Modulation for Enhanced Single-Cell Western Blotting.For whom the cells pull: Hydrogel and micropost devices for measuring traction forces.Hydrogels with tunable stress relaxation regulate stem cell fate and activity.The Predictive Link between Matrix and Metastasis.Biomaterials and emerging anticancer therapeutics: engineering the microenvironment.ITGA6 is directly regulated by hypoxia-inducible factors and enriches for cancer stem cell activity and invasion in metastatic breast cancer models.Obesity-dependent changes in interstitial ECM mechanics promote breast tumorigenesis.Strain-enhanced stress relaxation impacts nonlinear elasticity in collagen gels.A 3D in vitro model to explore the inter-conversion between epithelial and mesenchymal states during EMT and its reversion.Mechano-reciprocity is maintained between physiological boundaries by tuning signal flux through the Rho-associated protein kinase.A 3D Poly(ethylene glycol)-based Tumor Angiogenesis Model to Study the Influence of Vascular Cells on Lung Tumor Cell Behavior.Extracellular matrix stiffness causes systematic variations in proliferation and chemosensitivity in myeloid leukemias.Tumour-suppressor microRNAs regulate ovarian cancer cell physical properties and invasive behaviour.A novel double-targeted nondrug delivery system for targeting cancer stem cells.

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Extracellular matrix stiffness and composition jointly regulate the induction of malignant phenotypes in mammary epithelium.

http://www.wikidata.org/entity/Q38984674

description

2014 nî lūn-bûn

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2014年の論文

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2014年学术文章

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2014年学术文章

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2014年学术文章

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2014年学术文章

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2014年学术文章

@zh-sg

2014年學術文章

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2014年學術文章

@zh

2014年學術文章

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name

Extracellular matrix stiffness ...... notypes in mammary epithelium.

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type

label

Extracellular matrix stiffness ...... notypes in mammary epithelium.

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Extracellular matrix stiffness ...... notypes in mammary epithelium.

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Nature Materials

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Extracellular matrix stiffness ...... enotypes in mammary epithelium

@en

P2093

Catia S Verbeke

http://www.w3.org/2001/XMLSchema#string

Cristiana Branco da Cunha

http://www.w3.org/2001/XMLSchema#string

David J Mooney

http://www.w3.org/2001/XMLSchema#string

Jae-Won Shin

http://www.w3.org/2001/XMLSchema#string

Kimberly H Allison

http://www.w3.org/2001/XMLSchema#string

Ovijit Chaudhuri

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P2860

A mechanosensitive transcriptional mechanism that controls angiogenesis

Cell movement is guided by the rigidity of the substrate.

Integrin ligands at a glance

Matrix crosslinking forces tumor progression by enhancing integrin signaling

Matrix elasticity directs stem cell lineage specification

Tensional homeostasis and the malignant phenotype

Integrin signaling

Estrogen receptor alpha in human breast cancer: occurrence and significance

Tissue cells feel and respond to the stiffness of their substrate

Why don't we get more cancer? A proposed role of the microenvironment in restraining cancer progression

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10.1038/NMAT4009

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extracellular matrix