A pSMAD/CDX2 complex is essential for the intestinalization of epithelial metaplasia.
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Gastrointestinal stem cells in health and disease: from flies to humansTransformation of intestinal stem cells into gastric stem cells on loss of transcription factor Cdx2.BMP-driven NRF2 activation in esophageal basal cell differentiation and eosinophilic esophagitisBMP4 Signaling Is Able to Induce an Epithelial-Mesenchymal Transition-Like Phenotype in Barrett's Esophagus and Esophageal Adenocarcinoma through Induction of SNAIL2In oesophageal squamous cells, nitric oxide causes S-nitrosylation of Akt and blocks SOX2 (sex determining region Y-box 2) expressionBarrett's Esophagus: A Comprehensive and Contemporary Review for Pathologists.Comparison of newly developed anti-bone morphogenetic protein 4 llama-derived antibodies with commercially available BMP4 inhibitors.The characterization of an intestine-like genomic signature maintained during Barrett's-associated adenocarcinogenesis reveals an NR5A2-mediated promotion of cancer cell survival.Mechanisms of Barrett's oesophagus: intestinal differentiation, stem cells, and tissue models.Esophageal development and epithelial homeostasis.Barrett's metaplasia develops from cellular reprograming of esophageal squamous epithelium due to gastroesophageal reflux.Deoxycholic acid (DCA) confers an intestinal phenotype on esophageal squamous epithelium via induction of the stemness-associated reprogramming factors OCT4 and SOX2.Inactivation of NKX6.3 in the stomach leads to abnormal expression of CDX2 and SOX2 required for gastric-to-intestinal transdifferentiation.Contribution of immunomodulators to gastroesophageal reflux disease and its complications: stromal cells, interleukin 4, and adiponectin.Transcommitment: Paving the Way to Barrett's Metaplasia.The Barrett's Gland in Phenotype Space.Reflux esophagitis and its role in the pathogenesis of Barrett's metaplasia.MicroRNA Expression can be a Promising Strategy for the Detection of Barrett's Esophagus: A Pilot Study.Esophageal Cancer: Genomic and Molecular Characterization, Stem Cell Compartment and Clonal Evolution.Surgical Models of Gastroesophageal Reflux with MiceUEG Week 2015 Poster PresentationsSingle cell RNA-seq reveals profound transcriptional similarity between Barrett's oesophagus and oesophageal submucosal glands
P2860
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P2860
A pSMAD/CDX2 complex is essential for the intestinalization of epithelial metaplasia.
description
2014 nî lūn-bûn
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2014年の論文
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2014年論文
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2014年論文
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2014年論文
@zh-hk
2014年論文
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2014年論文
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2014年论文
@wuu
2014年论文
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2014年论文
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name
A pSMAD/CDX2 complex is essential for the intestinalization of epithelial metaplasia.
@en
type
label
A pSMAD/CDX2 complex is essential for the intestinalization of epithelial metaplasia.
@en
prefLabel
A pSMAD/CDX2 complex is essential for the intestinalization of epithelial metaplasia.
@en
P2093
P1433
P1476
A pSMAD/CDX2 complex is essential for the intestinalization of epithelial metaplasia
@en
P2093
Danielle Straub
Kaushal Parikh
Kausilia K Krishnadath
Kees K Hoeben
Navtej S Buttar
Paul Fockens
Vincent Everts
P304
P356
10.1016/J.CELREP.2014.03.074
P577
2014-05-01T00:00:00Z