Inflammation-induced NFATc1-STAT3 transcription complex promotes pancreatic cancer initiation by KrasG12D. - Wikidata - wikimedia - TriplyDB

Inflammation-induced NFATc1-STAT3 transcription complex promotes pancreatic cancer initiation by KrasG12D.

Inflammation-induced NFATc1-STAT3 transcription complex promotes pancreatic cancer initiation by KrasG12D.

http://www.wikidata.org/entity/Q39008845

about

Cell cycle and apoptosis regulation by NFAT transcription factors: new roles for an old player Paving the Road to Tumor Development and Spreading: Myeloid-Derived Suppressor Cells are Ruling the Fate Bidirectional interconversion of stem and non-stem cancer cell populations: A reassessment of theoretical models for tumor heterogeneity G-CSF Promotes Neuroblastoma Tumorigenicity and Metastasis via STAT3-Dependent Cancer Stem Cell Activation.CXCR2 Inhibition Profoundly Suppresses Metastases and Augments Immunotherapy in Pancreatic Ductal Adenocarcinoma.IL17 Functions through the Novel REG3β-JAK2-STAT3 Inflammatory Pathway to Promote the Transition from Chronic Pancreatitis to Pancreatic Cancer NFATc4 Regulates Sox9 Gene Expression in Acinar Cell Plasticity and Pancreatic Cancer Initiation Nuclear Factor of Activated T Cells-dependent Down-regulation of the Transcription Factor Glioma-associated Protein 1 (GLI1) Underlies the Growth Inhibitory Properties of Arachidonic Acid.Inhibiting NFAT1 for breast cancer therapy: New insights into the mechanism of action of MDM2 inhibitor JapA Idiopathic pancreatitis in a patient with a STAT3 mutation.A threshold level of NFATc1 activity facilitates thymocyte differentiation and opposes notch-driven leukaemia development GRIM19 ameliorates acute graft-versus-host disease (GVHD) by modulating Th17 and Treg cell balance through down-regulation of STAT3 and NF-AT activation.The pancreatitis-associated protein VMP1, a key regulator of inducible autophagy, promotes Kras(G12D)-mediated pancreatic cancer initiation.Plasma microRNA panels to diagnose pancreatic cancer: Results from a multicenter study Th17 cells in cancer: the ultimate identity crisis.Stromal biology and therapy in pancreatic cancer: a changing paradigm.STAT3-Interacting Proteins as Modulators of Transcription Factor Function: Implications to Targeted Cancer Therapy.GSK-3β Governs Inflammation-Induced NFATc2 Signaling Hubs to Promote Pancreatic Cancer Progression.Diagnostic value of selected markers and apoptotic pathways for pancreatic cancer.New insights in the development of pancreatic cancer.NFATc1 Links EGFR Signaling to Induction of Sox9 Transcription and Acinar-Ductal Transdifferentiation in the Pancreas.Antithetical NFATc1-Sox2 and p53-miR200 signaling networks govern pancreatic cancer cell plasticity NFATc1 controls the cytotoxicity of CD8+ T cells.NFATc1 as a therapeutic target in FLT3-ITD-positive AML.Phospho-valproic acid inhibits pancreatic cancer growth in mice: enhanced efficacy by its formulation in poly-(L)-lactic acid-poly(ethylene glycol) nanoparticles.Luteolin decreases invasiveness, deactivates STAT3 signaling, and reverses interleukin-6 induced epithelial-mesenchymal transition and matrix metalloproteinase secretion of pancreatic cancer cells.Store-Operated Ca2+ Entry Controls Clonal Expansion of T Cells through Metabolic Reprogramming.Induction of Short NFATc1/αA Isoform Interferes with Peripheral B Cell Differentiation.Interferon gamma, an important marker of response to immune checkpoint blockade in non-small cell lung cancer and melanoma patients.Role of the EZH2/miR-200 axis in STAT3-mediated OSCC invasion.The Transcription Factor NFATc1 Supports the Rejection of Heterotopic Heart Allografts.

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Inflammation-induced NFATc1-STAT3 transcription complex promotes pancreatic cancer initiation by KrasG12D.

http://www.wikidata.org/entity/Q39008845

description

2014 nî lūn-bûn

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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2014年论文

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2014年论文

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name

Inflammation-induced NFATc1-ST ...... cancer initiation by KrasG12D.

@en

Inflammation-induced NFATc1-ST ...... cancer initiation by KrasG12D.

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type

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Inflammation-induced NFATc1-ST ...... cancer initiation by KrasG12D.

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Inflammation-induced NFATc1-ST ...... cancer initiation by KrasG12D.

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Inflammation-induced NFATc1-ST ...... cancer initiation by KrasG12D.

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Inflammation-induced NFATc1-ST ...... cancer initiation by KrasG12D.

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Cancer Discovery

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Inflammation-induced NFATc1-ST ...... cancer initiation by KrasG12D.

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Albrecht Neesse

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Alexander König

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Daniel D Billadeau

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Elisabeth Heßmann

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Garima Singh

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Jens T Siveke

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Jin-San Zhang

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Johanna Reinecke

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Julius Nikorowitsch

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Marek Bartkuhn

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P2860

Genomic views of distant-acting enhancers

Gene set enrichment analysis: a knowledge-based approach for interpreting genome-wide expression profiles.

ENCODE whole-genome data in the UCSC genome browser (2011 update)

Histone H3K27ac separates active from poised enhancers and predicts developmental state

Inflammatory networks and immune surveillance of pancreatic carcinoma

Immunity, Inflammation, and Cancer

NF-AT components define a family of transcription factors targeted in T-cell activation

Transcription factors of the NFAT family: regulation and function

Novel interactions between NFATc1 (Nuclear Factor of Activated T cells c1) and STAT-3 (Signal Transducer and Activator of Transcription-3) mediate G protein-coupled receptor agonist, thrombin-induced biphasic expression of cyclin D1, with first phas

EGF receptor is required for KRAS-induced pancreatic tumorigenesis

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6

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261326276

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