The NADPH oxidase NOX4 inhibits hepatocyte proliferation and liver cancer progression.
about
Role of NADPH oxidases in the redox biology of liver fibrosisOxidative stress in cancer and fibrosis: Opportunity for therapeutic intervention with antioxidant compounds, enzymes, and nanoparticlesCRISPR-Cas9 Mediated NOX4 Knockout Inhibits Cell Proliferation and Invasion in HeLa CellsEuropean contribution to the study of ROS: A summary of the findings and prospects for the future from the COST action BM1203 (EU-ROS).Decoding NADPH oxidase 4 expression in human tumors.Overexpression of NOX4 predicts poor prognosis and promotes tumor progression in human colorectal cancer.The Role of NOX4 and TRX2 in Angiogenesis and Their Potential Cross-Talk.Oxidative stress and hepatic Nox proteins in chronic hepatitis C and hepatocellular carcinoma.Redox regulation of cardiomyocyte cell cycling via an ERK1/2 and c-Myc-dependent activation of cyclin D2 transcription{2-[1-(3-Methoxycarbonylmethyl-1H-indol-2-yl)-1-methyl-ethyl]-1H-indol-3-yl}-acetic Acid Methyl Ester Inhibited Hepatocellular Carcinoma Growth in Bel-7402 Cells and Its Resistant Variants by Activation of NOX4 and SIRT3.The NADPH oxidase NOX4 represses epithelial to amoeboid transition and efficient tumour dissemination.ROS and energy metabolism in cancer cells: alliance for fast growth.Biological Activities of Reactive Oxygen and Nitrogen Species: Oxidative Stress versus Signal Transduction.The human Nox4: gene, structure, physiological function and pathological significance.NADPH Oxidases in Chronic Liver DiseasesTherapeutic potential of NADPH oxidase 1/4 inhibitors.Dissecting the role of epidermal growth factor receptor catalytic activity during liver regeneration and hepatocarcinogenesis.NADPH Oxidase 1 and NADPH Oxidase 4 Have Opposite Prognostic Effects for Patients with Hepatocellular Carcinoma after Hepatectomy.Gene expression of NOX family members and their clinical significance in hepatocellular carcinoma.Oxidative Stress in Oral Diseases: Understanding Its Relation with Other Systemic Diseases.Mcl-1 regulates reactive oxygen species via NOX4 during chemotherapy-induced senescence.Gene silencing of Nox4 by CpG island methylation during hepatocarcinogenesis in rats.RNAi-mediated silencing of NOX4 inhibited the invasion of gastric cancer cells through JAK2/STAT3 signaling.NOX4-driven ROS formation mediates PTP inactivation and cell transformation in FLT3ITD-positive AML cells.Oxidative stress promotes myocardial fibrosis by upregulating KCa3.1 channel expression in AGT-REN double transgenic hypertensive mice.
P2860
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P2860
The NADPH oxidase NOX4 inhibits hepatocyte proliferation and liver cancer progression.
description
2014 nî lūn-bûn
@nan
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
2014年论文
@zh
2014年论文
@zh-cn
name
The NADPH oxidase NOX4 inhibits hepatocyte proliferation and liver cancer progression.
@en
The NADPH oxidase NOX4 inhibits hepatocyte proliferation and liver cancer progression.
@nl
type
label
The NADPH oxidase NOX4 inhibits hepatocyte proliferation and liver cancer progression.
@en
The NADPH oxidase NOX4 inhibits hepatocyte proliferation and liver cancer progression.
@nl
prefLabel
The NADPH oxidase NOX4 inhibits hepatocyte proliferation and liver cancer progression.
@en
The NADPH oxidase NOX4 inhibits hepatocyte proliferation and liver cancer progression.
@nl
P50
P1476
The NADPH oxidase NOX4 inhibits hepatocyte proliferation and liver cancer progression.
@en
P2093
Estanis Navarro
Joan Fernando
P304
P356
10.1016/J.FREERADBIOMED.2014.01.040
P50
P577
2014-02-06T00:00:00Z