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Gene expression profiling uncovers molecular classifiers for the recognition of anaplastic large-cell lymphoma within peripheral T-cell neoplasms.Identification of a 3-gene model as a powerful diagnostic tool for the recognition of ALK-negative anaplastic large-cell lymphoma.Functional validation of the anaplastic lymphoma kinase signature identifies CEBPB and BCL2A1 as critical target genes.MicroRNA expression profiling identifies molecular signatures associated with anaplastic large cell lymphomaTransposable elements: The enemies within.ALK-dependent control of hypoxia-inducible factors mediates tumor growth and metastasis.Molecular events underlying interleukin-6 independence in a subclone of the CMA-03 multiple myeloma cell line.STAT3-mediated activation of microRNA cluster 17~92 promotes proliferation and survival of ALK-positive anaplastic large cell lymphoma.The EGFR family members sustain the neoplastic phenotype of ALK+ lung adenocarcinoma via EGR1.Identification of a new subclass of ALK-negative ALCL expressing aberrant levels of ERBB4 transcripts.Ablation of oncogenic ALK is a viable therapeutic approach for anaplastic large-cell lymphomas.IRF4 Mediates the Oncogenic Effects of STAT3 in Anaplastic Large Cell Lymphomas.ALK expression favorably impacts the prognosis of NRAS-mutated metastatic melanomasIDH2 inhibition enhances proteasome inhibitor responsiveness in hematological malignancies
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P50
description
hulumtuese
@sq
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Elisa Pellegrino
@ast
Elisa Pellegrino
@en
Elisa Pellegrino
@es
Elisa Pellegrino
@nl
Elisa Pellegrino
@sl
type
label
Elisa Pellegrino
@ast
Elisa Pellegrino
@en
Elisa Pellegrino
@es
Elisa Pellegrino
@nl
Elisa Pellegrino
@sl
prefLabel
Elisa Pellegrino
@ast
Elisa Pellegrino
@en
Elisa Pellegrino
@es
Elisa Pellegrino
@nl
Elisa Pellegrino
@sl
P106
P1153
54680541800
P21
P31
P496
0000-0002-8629-8172